自由群的直积的检验元素的注记

本文研究了自由群的直积的检验元素,通过对直积的自同态的分解,得到了直积中的元素为检验元素的充分必要条件,改进了O'neill和Turner的结果.此外,构造了两类具体的检验元素.     

乘方的指数与其幂的位数的关系

为了探究乘方的指数与其幂的位数的关系,定义了几个有关的新概念,并且证明了两个关于乘方以及进制进位的定理,由此建立起关于乘方以及进制进位的理论体系,其中包括进位理论中判定乘方的指数与其幂的位数是否存在周期规律的判别法,以及进位规律的求解法和四条相关的性质.     

树的最大特征值的上界的一个注记

设T是一个树,V是T的顶点集．记dv是υ∈V的度,△是T的最大顶点度．设υ∈V且dw=1．记k=ew+1,这里ew是w的excentricity．设δj′= max{dυ:dist(υ,w)=j},j=1,2,…,k-2,我们证明和这里μ1(T)和λ1(T)分别是T的Laplacian矩阵和邻接矩阵的最大特征值．特别地,记δo′=2．     

Lindelf的工作与Chaplygin的工作的互补性

运用Vakonomic模型导出Lindel f方程 ,表明Lindel f的工作与Vakonomic模型相吻合 ;运用Chetaev模型导出Chaplygin方程 ,表明Chaplygin的工作与Chetaev模型相吻合·　在此基础上 ,通过改进Chaplygin方程和Lindel f方程的表示形式 ,实现了从Lindel f方程向Chaplygin方程的合理过渡和从Chaplygin方程向Lindel f方程的合理的过渡·　最后 ,给出一个典型实例·　结果表明 ,正如Vako nomic模型与Chetaev模型是互补的一样 ,Lindel f的工作与Chaplygin的工作也是互补的·     

抛物线上的点列构成的多边形的面积问题

引理设Pn,Pn＋d,Pn＋2d,…,Pn＋（k-1）d是抛物线y=ax^2＋vx＋c（a,b,c为常数,a≠0）上的点列（注：本文用只表示函数图象上横坐标为t的点）,则k边形Pn＋dPn＋2d…Pn＋（k-1）d的面积S与n无关（即与点列的起始位置无关）,     

有趣的圆的膨胀和收缩

向平静的湖面投入一颗石子,形成水波纹,这水波纹构成以投入点为圆心的一族同心圆,给我们以点膨胀为圆的最好形象.反之如图1,⊙O的半径OA=r,让⊙O的半径逐渐缩小,形成一族同心圆,当⊙O的半径r趋近于零时,⊙O收缩为一点O,因此,把点看成半径长为零的圆.     

计算Hamilton矩阵特征值的一个稳定的有效的保结构的算法

提出了一个稳定的有效的保结构的计算Hamilton矩阵特征值和特征不变子空间的算法,该算法是由SR算法改进变形而得到的。在该算法中,提出了两个策略,一个叫做消失稳策略,另一个称为预处理技术。在消失稳策略中,通过求解减比方程和回溯彻底克服了Bunser Gerstner和Mehrmann提出的SR算法的严重失稳和中断现象的发生,两种策略的实施的代价都非常低。数值算例展示了该算法比其它求解Hamilton矩阵特征问题的算法更有效和可靠。     

成功的门都是虚掩着的

在1968年墨西哥奥运会上,美国选手海因斯撞线后,转过身看着9.95秒的成绩牌,摊开双手自言自语地说了一句话.这一情景通过电视有好几亿人看到,可是由于当时他身边没有话筒,因此海因斯到底说了句什么话,谁都不知道.16年后,一位记者看墨西哥奥运会的资料片,当再次看到海因斯的镜头时,他想,这是人类历史上第一次有人在百米赛道上突破10秒大关,海因斯在看到记录的那一瞬间,一定说了一句不同凡响的话.这一新闻点,竟被那么多记者给漏掉了.于是他决定去采访海因斯,问海因斯当     

与一类算术函数关联的矩阵的行列式的下界

设f为一个算术函数,S={x 1,…,x n}为一个n元正整数集合.称S为gcd-封闭的, 如果对于任意1 i,j n,均有(x i,x j)∈S.以 ={y 1,…,y m}表示包含S的最小gcd-封闭的正整数集合. 设(f(x i,x j))表示一个n×n矩阵, 其(i,j)项为f在x i与x j的最大公因子(x i,x j)处的值. 设(f[x i,x j])表示一个n×n矩阵, 其(i,j)项为f在x i与x j的最小公倍数[x i.xj]处的值. 本文证明了: (i) 如果f∈C s ={f:(f*μ)(d)>0, x∈S,d|x},这里f*μ表示f与μ的Dirichlet乘积,μ表示M bius函数,那么 并且(1)取等号当且仅当S=;(ii)如果f为乘法函数,并且 ∈Cs,那么 并且(2)取等号当且仅当S= .不等式(1)和(2)分别改进了Bourque与Ligh在1993年和1995年所得到的结果.     

一类具有转向点的三阶微分方程的边值问题的解的高阶渐近展开

利用上下解方法,研究如下一类具有转向点的三阶微分方程的边值问题{ε~2y″′=f(t,y,ε)y″ g(t,y,ε)y′ h(t,y,ε),a相似文献   

计算机蠕虫病毒传播的数学模型分析

建立了一种计算机病毒传播的数学模型,并利用微分方程理论进行了分析,得到了计算机病毒消除的阈值,并进行了数值模拟.     

A bi-virus competing model with time-varying susceptibility and repeated infection

This paper introduces a bi-virus model with time-varying susceptibility. The model describes the case that there coexist two viruses and the time-varying susceptibility due to repeated infections. For different parameters, we investigate the stability of various equilibriums. Under appropriate conditions the two viruses show competitive relationship, that is, one virus will eventually become a pandemic, and the other virus will eventually disappear. For this case, we further study the dynamical behavior of virus transmission. The model shows some new phenomena, that is, the outbreak of the virus will be delayed appropriately, giving people an illusion. Finally, we present a numerical example to illustrate the effectiveness of the theoretical results.     

Global properties of an improved hepatitis B virus model

Hepatitis B virus (HBV) infection is an important health problem worldwide. In this paper, we introduce an improved HBV model with standard incidence function and cytokine-mediated ‘cure’ based on empirical evidences. By carrying out a global analysis of the modified model and studying the stability of the equilibria, we show that infection-free equilibrium is globally asymptotically stable if the basic reproduction number of virus is less than one and, conversely, the infection equilibrium is globally asymptotically stable if the basic reproduction number of virus is greater than one. The study and information derived from this model and other related models may have an important impact on preventing mortality due to hepatitis B virus in the future.     

Mathematical analysis of a virus dynamics model with general incidence rate and cure rate

The rate of infection in many virus dynamics models is assumed to be bilinear in the virus and uninfected target cells. In this paper, the dynamical behavior of a virus dynamics model with general incidence rate and cure rate is studied. Global dynamics of the model is established. We prove that the virus is cleared and the disease dies out if the basic reproduction number R₀≤1 while the virus persists in the host and the infection becomes endemic if R₀>1.     

A with-in host Dengue infection model with immune response

A model of viral infection of monocytes population by Dengue virus is formulated here. The model can capture phenomena that dengue virus is quickly cleared in approximately 7 days after the onset of the symptoms. The model takes into account the immune response. It is shown that the quantity of free virus is decreasing when the viral invasion rate is increasing. The basic reproduction ratio of model without immune response is reduced significantly by adding the immune response. Numerical simulations indicate that the growth of immune response and the invasion rate are very crucial in identification of the intensity of infection.     

病毒感染群体动力学模型分析

本文构建并讨论了一类病毒感染的群体动力学模型,得到了模型存在轨道稳定周 期解的充分条件,较好地解释了在病毒持续感染者体内观察到的病毒载量波动现象.     

带有免疫反应的病毒动力学模型的全局稳定性

利用Lyapunov函数研究了带有免疫反应的病毒动力学模型的全局稳定性.当基本再生数R0≤1时.病毒在体内清除;当R0>1时,病毒在体内持续生存.并且模型的正解当免疫再生数R1≤1时,趋于无免疫平衡点,当R1>1.趋于地方病平衡点.     

Dynamical behaviors of a discrete HIV‐1 virus model with bilinear infective rate

We establish a discrete virus dynamic model by discretizing a continuous HIV‐1 virus model with bilinear infective rate using ‘hybrid’ Euler method. We discuss not only the existence and global stability of the uninfected equilibrium but also the existence and local stability of the infected equilibrium. We prove that there exists a crucial value similar to that of the continuous HIV‐1 virus dynamics, which is called the basic reproductive ratio of the virus. If the basic reproductive ratio of the virus is less than one, the uninfected equilibrium is globally asymptotically stable. If the basic reproductive ratio of the virus is larger than one, the infected equilibrium exists and is locally stable. Moreover, we consider the permanence for such a system by constructing a Lyapunov function v_n. Copyright © 2013 John Wiley & Sons, Ltd.     

Dynamics of an HIV‐1 virus model with both virus‐to‐cell and cell‐to‐cell transmissions,general incidence rate,intracellular delay,and CTL immune responses

Direct cell‐to‐cell transmission of HIV‐1 is a more efficient means of virus infection than virus‐to‐cell transmission. In this paper, we incorporate both these transmissions into an HIV‐1 virus model with nonlinear general incidence rate, intracellular delay, and cytotoxic T lymphocyte (CTL) immune responses. This model admits three types of equilibria: infection‐free equilibrium, CTL‐inactivated equilibrium, and CTL‐activated equilibrium. By using Lyapunov functionals and LaSalle invariance principle, it is verified that global threshold dynamics of the model can be explicitly described by the basic reproduction numbers.     

Dynamics of an HIV-1 infection model with cell mediated immunity

In this paper, we study the dynamics of an improved mathematical model on HIV-1 virus with cell mediated immunity. This new 5-dimensional model is based on the combination of a basic 3-dimensional HIV-1 model and a 4-dimensional immunity response model, which more realistically describes dynamics between the uninfected cells, infected cells, virus, the CTL response cells and CTL effector cells. Our 5-dimensional model may be reduced to the 4-dimensional model by applying a quasi-steady state assumption on the variable of virus. However, it is shown in this paper that virus is necessary to be involved in the modeling, and that a quasi-steady state assumption should be applied carefully, which may miss some important dynamical behavior of the system. Detailed bifurcation analysis is given to show that the system has three equilibrium solutions, namely the infection-free equilibrium, the infectious equilibrium without CTL, and the infectious equilibrium with CTL, and a series of bifurcations including two transcritical bifurcations and one or two possible Hopf bifurcations occur from these three equilibria as the basic reproduction number is varied. The mathematical methods applied in this paper include characteristic equations, Routh–Hurwitz condition, fluctuation lemma, Lyapunov function and computation of normal forms. Numerical simulation is also presented to demonstrate the applicability of the theoretical predictions.