具有染病者输入的离散SIR传染病模型分析

引入相应的概率建立了具有染病者输入的离散SIR传染病模型,确定了决定其动力学性态的阂值.在阈值之下模型仅存在无病平衡点,且无病平衡点是全局渐近稳定的;在阈值之上模型是一致持续的,有唯一的地方病平衡点存在,且地方病平衡点是局部渐近稳定的.     

一类带有一般接触率和常数输入的流行病模型的全局分析

借助极限系统理论和构造适当的Liapunov函数,对带有一般接触率和常数输入的SIR型和SIRS型传染病模型进行讨论．当无染病者输入时,地方病平衡点存在的阈值被找到A·D2对相应的SIR模型,关于无病平衡点和地方病平衡点的全局渐近稳定性均得到充要条件;对相应的SIRS模型,得到无病平衡点和地方病平衡点全局渐近稳定的充分条件．当有染病者输入时,模型不存在无病平衡点．对相应的SIR模型,地方病平衡点是全局渐近稳定的;对相应的SIRS模型,得到地方病平衡点全局渐近稳定的充分条件．     

一类具有时滞的流行病模型分析

讨论了一类带有时滞的SE IS流行病模型,并讨论了阈值、平衡点和稳定性.模型是一个具有确定潜伏期的时滞微分方程模型,在这里我们得到了各类平衡点存在条件的阈值R0;当R0<1时,只有无病平衡点P0,且是全局渐近稳定的;当R0>1时,除无病平衡点外还存在唯一的地方病平衡点Pe,且该平衡点是绝对稳定的.     

一类带有接种的流行病模型的全局稳定性

该文讨论了一类带有接种的流行病模型. 在该模型中假设恢复后的个体与被接种的个体均具有确定的免疫期, 它是一个时滞微分系统. 通过分析, 得到了地方病平衡点存在的阈值, 以及无病平衡点和地方病平衡点局部渐近稳定和全局渐近稳定的充分条件.     

具有标准发生率和因病死亡率的离散SIS传染病模型的全局稳定性分析

主要研究了具有标准发生率和因病死亡率的离散SIS传染病模型的动力学性质,利用构造Lyapunov函数,得到模型无病平衡点和地方性平衡点的全局稳定性,即无病平衡点是全局渐近稳定的当且仅当基本再生数R_0≤1,地方病平衡点是全局渐近稳定的当且仅当R_0>1.     

垂直传染与环境因素对野牛布鲁氏菌传播的影响研究

研究了布鲁氏菌通过水平和垂直传染在野牛种群中传播的非线性动态模型.在SIR模型中引入了环境中的布鲁氏菌对野牛的影响,并提出了一种SIRB模型.分别算出了该模型的无病平衡点P_0和地方病平衡点P*,利用再生矩阵得到模型的阈值R_0,证明了模型平衡点的稳定性由阈值的大小所决定,即R_0 1时,通过构造合适的Lyapunov函数,证得无病平衡点全局渐近稳定.当R_0 1时,利用几何方法,证得地方病平衡点全局渐近稳定.     

一类非线性染病年龄结构模型渐近性态

研究一类具有非线性染病年龄结构SIS流行病传播数学模型动力学性态,得到疾病绝灭和持续生存的阈值--基本再生数.当基本再生数小于或等于1时,仅存在无病平衡点,且在其小于1的情况下,无病平衡点全局渐近稳定,疾病将逐渐消除;当基本再生数大于1时,存在不稳定的无病平衡点和唯一的局部渐近稳定的地方病平衡点,疾病将持续存在.     

考虑部分免疫和环境传播的麻疹传染病模型的全局稳定性

该文考虑一个具有部分免疫和环境传播的麻疹传染病模型,得到基本再生数R_0,并通过构造Lyapunov函数,研究了该模型的无病平衡点和地方病平衡点的全局稳定性.当R_01时,无病平衡点是全局渐近稳定的,即麻疹不会传播开;当R_01时,模型存在唯一的地方病平衡点,且是全局渐近稳定的,即麻疹的传播保持在一个稳定的状态.最后,通过数值分析说明了这些结果的合理性.该文工作对于预防和控制麻疹病毒的传播具有实际意义.     

一年龄结构乙肝传染病模型及稳定性

本文讨论一年龄结构的乙肝传染病模型,得到基本再生数■的表达式,证明当■时,无病平衡点局部渐近稳定且全局渐近稳定;当■时,存在唯一的地方病平衡点,并给出地方病平衡点的局部渐近稳定性条件,这些条件对于控制疾病的传播具有重要的理论及实际意义.     

一类潜伏期和染病期均传染的SEIQR模型的全局稳定性

研究一类潜伏期和染病期均传染的SEIQR传染病模型,得到疾病流行与否的阈值R_0.运用Lyapunov函数方法、LaSalle不变性原理及第二加性复合矩阵理论证明了当R_0≤1时无病平衡点全局渐近稳定,当R_01时地方病平衡点全局渐近稳定.     

一类具免疫应答和非线性感染函数的时滞HIV-1感染模型的全局稳定性

考虑到HIV-1感染过程中免疫反应和非线性感染函数,建立了一类具有三个分布时滞的HIV-1感染动力学模型.得到了关于病毒感染的基本再生数R0和CTLs免疫反应的基本再生数R1 ＜R0.通过构造Lyapunov泛函证明了系统具有阈值动力学性质,即当R0≤1时,系统存在全局渐近稳定的无感染平衡点;当R1≤1＜R0时,系统出...     

Global analysis of a dynamical model for transmission of tuberculosis with a general contact rate

This paper deals with the global analysis of a dynamical model for the spread of tuberculosis with a general contact rate. The model exhibits the traditional threshold behavior. We prove that when the basic reproduction ratio is less than unity, then the disease-free equilibrium is globally asymptotically stable and when the basic reproduction ratio is great than unity, a unique endemic equilibrium exists and is globally asymptotically stable under certain conditions. The stability of equilibria is derived through the use of Lyapunov stability theory and LaSalle’s invariant set theorem. Numerical simulations are provided to illustrate the theoretical results.     

GLOBAL STABILITY OF AN SIRS EPIDEMIC MODEL WITH DELAYS

In this article, an SIRS epidemic model spread by vectors (mosquitoes) which have an incubation time to become infectious is formulated. It is shown that a disease-free equilibrium point is globally stable if no endemic equilibrium point exists. Further, the endemic equilibrium point (if it exists) is globally stable with a respect "weak delay". Some known results are generalized.     

一类采取隔离措施的非线性传染率传染病模型的全局稳定性

讨论一类采取隔离措施的非线性传染率传染病的数学模型,得到了基本再生数Rθ的表达式,当Rθ<1时,仅存在无病平衡点,是全局渐近稳定的;当Rθ>1时,存在两个平衡点,其中无病平衡点不稳定,地方病平衡点全局渐近稳定.     

Analysis of epidemic spreading of an SIRS model in complex heterogeneous networks

In this paper, we study the spreading of infections in complex heterogeneous networks based on an SIRS epidemic model with birth and death rates. We find that the dynamics of the network-based SIRS model is completely determined by a threshold value. If the value is less than or equal to one, then the disease-free equilibrium is globally attractive and the disease dies out. Otherwise, the disease-free equilibrium becomes unstable and in the meantime there exists uniquely an endemic equilibrium which is globally asymptotically stable. A series of numerical experiments are given to illustrate the theoretical results. We also consider the SIRS model in the clustered scale-free networks to examine the effect of network community structure on the epidemic dynamics.     

Modeling and analysis of the spread of computer virus

Based on a set of reasonable assumptions, we propose a novel dynamical model describing the spread of computer virus. Through qualitative analysis, we give a threshold and prove that (1) the infection-free equilibrium is globally asymptotically stable if the threshold is less than one, implying that the virus would eventually die out, and (2) the infection equilibrium is globally asymptotically stable if the threshold is greater than one. Two numerical examples are presented to demonstrate the analytical results.     

两类带有确定潜伏期的SEIS传染病模型的分析

通过研究两类带有确定潜伏期的SEIS传染病模型,发现对种群的常数输入和指数输入会使疾病的传播过程产生本质的差异．对于带有常数输入的情形,找到了地方病平衡点存在及局部渐近稳定的阈值,证明了地方病平衡点存在时一定局部渐近稳定,并且疾病一致持续存在．对于带有指数输入的情形,发现地方病平衡点当潜伏期充分小时是局部渐近稳定的,当潜伏期充分大时是不稳定的．     

一类具有垂直传染和预防接种的SEIR传染病模型的定性分析

建立并分析了一类对出生时没有被染病母体垂直传染的染病者的新生儿进行免疫接种的SEIR传染病模型.得到了疾病是否灭绝的阈值R0,当R0<1时,无病平衡点全局渐近稳定的.当R0>1时,地方病平衡点局部渐近稳定的,且疾病一致持续生存.     

Global dynamics of a viral infection model with a latent period and Beddington-DeAngelis response

In this paper, we study the global dynamics of a viral infection model with a latent period. The model has a nonlinear function which denotes the incidence rate of the virus infection in vivo. The basic reproduction number of the virus is identified and it is shown that the uninfected equilibrium is globally asymptotically stable if the basic reproduction number is equal to or less than unity. Moreover, the virus and infected cells eventually persist and there exists a unique infected equilibrium which is globally asymptotically stable if the basic reproduction number is greater than unity. The basic reproduction number determines the equilibrium that is globally asymptotically stable, even if there is a time delay in the infection.