基于白噪声的网络传染病模型动力学分析北大核心CSCD

该文基于确定性网络传染病模型,建立了白噪声影响下的随机网络传染病模型,证明了模型全局解的存在唯一性,利用随机微分方程理论得到了传染病随机灭绝和随机持久的充分条件.结果表明,白噪声对网络传染病传播动力学有很大的影响,白噪声能有效抑制传染病的传播,大的白噪声甚至能让原本持久的传染病变得灭绝.最后,通过数值模拟验证了理论结果.     

按时滞转化的阶段结构SIS传染病模型

对一类按时滞转化的具有两个阶段结构的SIS传染病模型进行了分析,得到了传染病最终消除和成为地方病的阈值.即当传染率小于该阀值时,传染病最终消除;反之,此种传染病将成为地方病.     

具有非线性感染力的一类SIR传染病模型的阈值分析

文章针对一类SIR传染病模型进行了改进,考虑了非线性感染力对阈值的影响.主要分四种情形对非线性感染力下的传染病阈值进行了计算与分析.对结果分析可知,传染病的传播阈值与非线性感染力有着密切关系,同时,免疫率μ对传染病阈值λ_c也起着非常关键的决定性作用.     

带有非线性传染率的具有阶段结构的SI传染病模型

对带有非线性传染率的具有阶段结构的SI传染病模型进行了讨论,得到了传染病最终消除和成为地方病的条件.     

传染病模型的研究及应用

分析了传染病的传播扩散特点,建立了传染病传播扩散的微分方程模型.利用最大似然估计法对模型中的参数进行了估计.并以SARS传染扩散为例,利用网上的公开数据对模型进行了检验,所得结果与实际情况一致.此模型为传染病的预防和控制提供了理论依据.     

基于网络媒体信息的传染病传播模型及其仿真研究

当一种突发传染病开始流行时,政府、媒体会以各种形式告知民众,有防范意识的民众将采取一定的防范措施来降低感染率.考虑面对一种突发传染病,将易感群体划分为具有防范意识和不具有防范意识两种群体,利用生命周期理论,分析网络媒体信息报道对传染病传播的影响,以此为依据建立一种改进的传染病传播模型(MSI).利用网络大数据得到对传染病有防范意识群体的观测值信息,利用神经网络技术对模型MSI的参数进行反演.然后对模型MSI数值仿真得到传染病传播过程,提出了相应的控制措施.     

一类具有时滞传染病模型的稳定性

讨论了具有双时滞的SIS传染病模型.研究了一个边界平衡点的全局稳定性和正平衡点的局部稳定性,得到了传染病最终消失和成为地方病的阈值.     

基于灰色微分方程的传染病预测

本文归纳总结了传染病的几类预测方法,并通过一个例子,将灰色模型和微分方程模型结合,用于传染病的预测.     

一类传染病持续生存和消亡的阈值

利用齐次向量场与其诱导向量场的关系对一类传染病模型进行了进一步研究,讨论了其平衡点的存在性和稳定性,求出了该类传染病持续生存和最终消亡的阈值.     

传播动力学模型回顾与展望

传播动力学模型主要包括传染病动力学模型、计算机病毒传播模型以及谣言传播模型.首先从传染病动力学模型入手,分别介绍均匀混合传染病动力学模型和网络动力学模型;然后介绍计算机病毒传播模型以及谣言传播模型,并与传染病模型进行对比.最后作了总结与展望.     

具有阶段结构的SI传染病模型

本文对一类具有两个阶段结构的SI传染病模型进行了分析，得到了传染病最终消除和成为地方病的阈值。     

Spreading of epidemics on scale-free networks with nonlinear infectivity

In this paper, we study the spreading of epidemics on scale-free networks with infectivity which is nonlinear in the connectivity of nodes. We will show that the nonlinear infectivity is more appropriate than constant or linear ones, and give the epidemic threshold of the SIS model on a scale-free network with nonlinear infectivity. In addition, we compare the effects of nonlinear infectivity on the epidemic threshold with two other cases on infinite and finite scale-free networks, and find some new results, such as: with unit recovery rate and nonlinear irrational infectivity, the epidemic threshold is always positive; and the epidemic threshold can increase with network size on finite networks, contrary to the findings in all previous work.     

Competing spreading processes and immunization in multiplex networks

Epidemic spreading on physical contact network will naturally introduce the human awareness information diffusion on virtual contact network, and the awareness diffusion will in turn depress the epidemic spreading, thus forming the competing spreading processes of epidemic and awareness in a multiplex networks. In this paper, we study the competing dynamics of epidemic and awareness, both of which follow the SIR process, in a two-layer networks based on microscopic Markov chain approach and numerical simulations. We find that strong capacities of awareness diffusion and self-protection of individuals could lead to a much higher epidemic threshold and a smaller outbreak size. However, the self-awareness of individuals has no obvious effect on the epidemic threshold and outbreak size. In addition, the immunization of the physical contact network under the interplay between of epidemic and awareness spreading is also investigated. The targeted immunization is found performs much better than random immunization, and the awareness diffusion could reduce the immunization threshold for both type of random and targeted immunization significantly.     

Threshold behaviour of a stochastic SIR model

In this paper, we investigate the threshold behaviour of a susceptible-infected-recovered (SIR) epidemic model with stochastic perturbation. When the noise is small, we show that the threshold determines the extinction and persistence of the epidemic. Compared with the corresponding deterministic system, this value is affected by white noise, which is less than the basic reproduction number of the deterministic system. On the other hand, we obtain that the large noise will also suppress the epidemic to prevail, which never happens in the deterministic system. These results are illustrated by computer simulations.     

The threshold of a stochastic SIRS epidemic model with saturated incidence

In this paper, we investigate the dynamics of a stochastic SIRS epidemic model with saturated incidence. When the noise is small, we obtain a threshold of the stochastic system which determines the extinction and persistence of the epidemic. Besides, we find that large noise will suppress the epidemic from prevailing.     

一类具有阶段结构和接种的SIR传染病模型

根据不同程度的感染者有不同的传染率,建立了一个具有阶段结构和双线性传染率的S IR流行病模型,得到了模型的阈值参数R0,证明了模型平衡点的全局性态完全由R0的值确定.并进行了数值模拟.     

Threshold dynamics in a stochastic SIRS epidemic model with nonlinear incidence rate

We discuss the dynamic of a stochastic Susceptible-Infectious-Recovered-Susceptible (SIRS) epidemic model with nonlinear incidence rate.The crucial threshold $\tilde{R}_0$ is identified and this will determine the extinction and persistence of the epidemic when the noise is small. We also discuss the asymptotic behavior of the stochastic model around the endemic equilibrium of the corresponding deterministic system. When the noise is large, we find that a large noise intensity has the effect of suppressing the epidemic, so that it dies out. Finally, these results are illustrated by computer simulations.     

Assessing the effects of homosexuals and bisexuals on the intrinsic dynamics of HIV/AIDS in heterosexual settings

A deterministic compartmental sex-structured HIV/AIDS model for assessing the effects of homosexuals and bisexuals on the intrinsic dynamics of the disease in heterosexual settings in which homosexuality and bisexuality issues have remained taboo is presented. The epidemic threshold and equilibria for the model are determined and stabilities are investigated. Comprehensive qualitative analysis of the model including invariance of solutions and permanence are carried out. The epidemic threshold known as the basic reproductive number suggests that heterosexuality, homosexuality, and bisexuality influence the growth of the epidemic in HIV/AIDS affected populations and the partial reproductive number (homosexuality induced or heterosexuality and bisexuality induced) with the larger value influences the overall dynamics of the epidemic in a setting. Numerical simulations of the model show that as long as one of the partial reproductive numbers is greater than unity, the disease will exist in the population. We conclude from the study that homosexuality and bisexuality enlarge the epidemic in a heterosexual setting. The theoretical study highlights the need to carry out substantial research to map homosexuals and bisexuals as it has remained unclear as to what extent this group has contributed to the epidemic in heterosexual settings especially in southern Africa, which has remained the epidemiological locus of the epidemic.