带有分布时滞和非线性发生率的媒介-宿主传染病模型全局稳定性(英文)

本文分析一类带有分布时滞和非线性发生率的媒介-宿主传染病动力学性质,得到模型基本再生数R0,发现系统中平衡态的全局动力学性质能够由基本再生数来完全确定:即,如果R01,无病平衡态是全局渐近稳定的;如果R01,则系统存在唯一地方病平衡态,并且该平衡态是全局渐近稳定的.     

一类具有两阶段结构的自治SIS传染病系统

讨论了具有两阶段结构的自治SIS传染病系统,证明了该系统的边界平衡态和正平衡态的全局渐近稳定性,得到了使其渐近稳定的阈值.     

一类含有非线性传染率的传染病模型的全局稳定性

讨论了一类带有非线性传染率的SIRS型传染病模型,得到了无病平衡点和地方病平衡点存在的阈值条件,借助构造Dulac函数和Liapunov函数,找到了两类平衡点全局渐近稳定的充要条件.     

带有扩散和Beddington-DeAngelis响应函数的捕食模型的正平衡态

讨论了带有扩散和Beddington-DeAngelis响应函数的捕食模型在齐次Dirichlet边界条件下的正平衡态.给出了正平衡态存在的一个充要条件,当猎物种群相互干扰强的时候,得到正平衡态的稳定性与唯一性.     

带有脉冲免疫和传染年龄的传染病模型的全局吸引性

讨论了带有脉冲免疫和传染年龄的传染病模型.传染类的恢复率是传染年龄的函数,当染病再生数小于1时,文章得到无病周期解是全局吸引的.如果总人口规模变化,也可得到类似的结论.最后,提出了带有脉冲免疫和传染年龄传染病模型待解决的问题.     

一类具有空间异质和扩散的SVIR模型的全局稳定性

考虑了一类具有空间异质和反应扩散的SVIR传染病模型.当基本再生数等于1时,假定扩散系数为常数,证明了系统的无病平衡态是全局渐近稳定的.     

一类具有时滞和空间扩散的SIR传染病模型的行波解

研究了一类具有时滞和空间扩散的SIR传染病模型,通过分析相应的特征方程,讨论了系统每个平衡态的局部稳定性,通过运用交叉迭代方法和Schauder不动点定理,把行波解的存在性转化为一对上下解的存在性,通过构造一对上下解,得到了连接无病平衡态和地方病平衡态的行波解的存在性.     

扩散引起的一类捕食-食饵模型的不稳定性分析

针对一类具有HollingⅡ型功能捕获函数的捕食-食饵模型的扩散问题进行了研究,得到了无扩散时正平衡点的稳定条件,以及扩散存在时对正平衡点稳定性产生的影响,最后证明了无扩散时系统在第一象限存在极限环的问题.研究结果表明:在捕食-食饵模型中,扩散是使无扩散时稳定的平衡态向不稳定的平衡态转变的系统内动力,是生物模式形成的基础.     

一类捕食——食饵模型正平衡解的整体分歧

讨论了一类改进的Leslie-Gower和Holling-Type Ⅱ型捕食-食饵模型对应的平衡态系统正解的结构.以捕食者的出生率b为分歧参数,利用局部分歧理论和整体分歧理论,得到了此平衡态系统正解的存在性与参数b的关系,即当b适当大时,该平衡态系统具有共存正解.     

年龄结构乙肝传染病模型及稳定性

本文讨论一年龄结构乙肝传染病模型,得出基本再生数■的表达式,证明:当■1时,无病平衡态局部渐近稳定且全局渐近稳定;当■ 1时,存在唯一的地方病平衡态,并给出地方病平衡态的局部渐近稳定性条件,这些条件对于控制疾病的传播具有重要的理论及实际意义.     

复合型脆断的应变能判据

本文提出一个十分简单的复合型脆断判据,即应变能判据。该判据可以表示成:(K_Ⅰ/K_Ⅰc)2+(K_Ⅱ/K_ⅡC)2+(K_Ⅲ/K_ⅢC)2=1,它与文献中的实验数据非常一致,是一个实用的判据。本文还提出一个经验判据:(K_Ⅰ/K_Ⅰc)m+(K_Ⅱ/K_ⅡC)n=1,1≤≤2。     

平面应变和反平面应变复合型裂纹尖端的理想塑性应力场

在裂纹尖端的理想塑性应力分量都只是θ的函数的条件下.利用平衡方程.应力应变率关系、相容方程和屈服条件,本文导出了平面应变和反平面应变复合型裂纹尖端的理想塑性应力场的一般解析表达式.将这些一般解析表达式用于复合型裂纹.我们就可以得到Ⅰ-Ⅲ、Ⅱ-Ⅲ及Ⅰ-Ⅱ-Ⅲ复合型裂纹尖端的理想塑性应力场的解析表达式.     

Stability analysis of delay differential equation models of HIV-1 therapy for fighting a virus with another virus

Considering two kinds of delays accounting, respectively, for (i) a latent period between the time target cells are contacted by the virus particles and the time the virions enter the cells and (ii) a virus production period for new virions to be produced within and released from the infected cells, we develop and analyze a mathematical model for HIV-1 therapy by fighting a virus with another virus. For the different values of the basic reproduction number and the second basic reproduction number, we investigate the stability of the infection-free equilibrium, the single-infection equilibrium and the double-infection equilibrium. We conclude that increasing delays will decrease the values of the basic reproduction number and the second basic reproduction number. Our results have potential applications in HIV-1 therapy. The approach we use here is a combination of analysis of characteristic equations, Fluctuation Lemma and Lyapunov function.     

Dynamics of a two-strain vaccination model for polio

A new deterministic model for the transmission dynamics of two strains of polio, the vaccine-derived polio virus (VDPV) and the wild polio virus (WPV), in a population is designed and rigorously analysed. It is shown that Oral Polio Vaccine (OPV) reversion (leading to increased incidences of WPV and VDPV strains), together with the combined effect of vaccinating a fraction of the unvaccinated susceptible and missed susceptible children, could induce the phenomenon of backward bifurcation when the associated reproduction number of the model is less than unity. Furthermore, the model undergoes competitive exclusion, where the strain with the higher reproduction number (greater than unity) drives the other (with reproduction number less than unity) to extinction. In the absence of OPV reversions (leading to the co-existence of both strains in the population), it is shown that the disease-free equilibrium of the model is globally-asymptotically stable whenever the associated reproduction number is less than unity. Numerical simulations of the model suggest that the model undergoes the phenomenon of competitive exclusion, where the strain with the higher reproduction number (greater than unity) drives the other to extinction. Furthermore, co-existence of the two strains is feasible if their respective reproduction number are equal or approximately equal (and greater than unity).     

最小多项式矩阵与线性多变量系统(Ⅱ)

本文之(Ⅰ)[8]是关于最小多项式矩阵的理论;其(Ⅱ)是关于这一理论在线性多变量系统中的应用.在本部分的第一节中,我们利用(Ⅰ)中的理论,详细地讨论线性多变量系统输入问题的一些结果.在第二节中,利用对偶性,我们给出行n.p.m.及行生成组等概念,并讨论线性多变量系统输出问题的某些结果.在第三节中.我们讨论化状态空间型为多项式矩阵型的方法.在第四节中,我们讨论这一问题的反问题,即化多项式矩阵型为状态空间型的问题.为说明这些理论和方法,我们给出一些有趣的例子.     

Mathematical analysis of the dynamics of visceral leishmaniasis in the Sudan

In this paper we develop a mathematical model to study the dynamics of visceral leishmaniasis in the Sudan. To develop this model we consider the dynamics of the disease between three different populations, human, reservoir and vector populations. The model is analyzed at equilibrium and the stability of the equilibria is analyzed. The basic reproduction number is derived, and the threshold conditions for disease elimination established. Results show that the disease can be eliminated under certain conditions. Simulations of the model show that human treatment helps in disease control, and its synergy with vector control will more likely result in the elimination of the disease.     

一类具有二次感染和接种的两病株流行病模型

本文考虑了一类具有二次感染和接种的两病株流行病模型，通过定义每一病株的基本再生数和侵入再生数，我们分析了非负平衡态的稳定性并获得了这样结论：对于较低的接种水平，病株一感染者处于支配地位而病株二感染者将从易感人群中消失，对于非常高的接种水平，疾病将均被消除。     

The asymptotic profile of a dengue model on a growing domain driven by climate change

Global warming results in a slow expansion of habitat range of mosquitoes, an important vector of dengue virus. To understand the impact of this changing environment on the transmission of dengue virus, we develop a dengue model on a growing domain under the framework of reaction diffusion equations. By overcoming some difficulties of dynamical behaviors caused by diffusion terms with variable-dependent coefficients, we investigate the stabilities of the disease-free and endemic equilibria in terms of the associated basic reproduction number. Comparing our dengue model on a growing domain to the model on a fixed domain in terms of the basic reproduction number, we conclude that habitat expansion resulting from global warming catalyzes the spread of dengue fever, and it is negative to the control of dengue fever. Finally, numerical simulations are performed and show a good agreement with our analytical results.     

基于应变的Ⅰ/Ⅱ/Ⅲ复合型断裂准则

将已有的适用于平面断裂的最大周向应变（MTSN）准则,推广到适用于空间三维断裂的断裂准则.并具体讨论了Poisson(泊松)比对复合型断裂的面内断裂角与面外断裂角及断裂包络图的影响.Ⅰ/Ⅲ复合型断裂时,面外断裂角与Poisson比无关.Ⅱ/Ⅲ及Ⅰ/Ⅱ/Ⅲ复合型断裂条件下,面内断裂角随着Poisson比的增大而减小,面外断裂角随着Poisson比的增大而增大.在复合型断裂条件下,包络图均随着Poisson比增大而减小.且Poisson比对断裂包络图的影响大于面内断裂角,对面外断裂角影响最小.将本准则理论预测值与多组实验数据进行对比,预测值与实验值吻合较好,可知推广的MTSN准则能够较好地预测三维断裂.     

SWEIA艾滋病毒传染模型及应用

人类免疫缺陷病毒(HIV)是一种严重威胁生命的病毒,感染艾滋病毒患者一般经历四个阶段:i)艾滋病毒阴性的窗口期(W);ii)阳性的无症状潜伏期(E);iii)有症状期(Ⅰ);以及iv)移除阶段(A).为深入研究艾滋病传播过程,建立SWEIA艾滋病毒传染模型,定义基本再生数,分析无病与地方病平衡点的存在性和局部稳定性,根据2004至2015年中国艾滋病患者数据,采用遗传算法对SWEIA模型中参数进行估计.通过对基本再生数敏感性分析以及模型数值随参数不同而产生的变化,揭示艾滋病窗口期的接触率是影响艾滋病流行的主要原因之一.