一类具有Allee影响的捕食与被捕食模型

分析并建立了具有Allee影响的捕食与被捕食模型,被捕食者由于自身繁殖或是被捕食而具有了Allee效应,分别讨论了强Allee和弱Allee对被捕食种群的影响,讨论了解的有界性和各平衡点的存在性,并证明了各平衡点的局部渐近稳定性,进一步通过构造适当的Lyapunov函数分析了正平衡点E*的全局渐近稳定性.     

一个带有Holling-Ⅳ功能性反应的捕食与被捕食模型的稳定性和分支

研究了一个带Holling-Ⅳ型功能反应的捕食与被捕食模型,讨论了系统解的有界性和各平衡点的存在性,使用Routh-Hurwitz定理得到了平衡点局部渐近稳定的充分条件.引入两个离散时滞,得出了重要的结果:边界平衡点的稳定性随着τ1的增加,由稳定变为不稳定,并且会发生Hopf分支.对正平衡点的稳定性变化,考虑了两个时滞相等的情况,结果是随着分支参数的增加,不仅稳定性会发生变化,产生Hopf分支,甚至可能出现小范围周期解.     

一类具有时滞的疾病感染的捕食-被捕食模型分析

研究了一类具有时滞的疾病感染的捕食-被捕食模型.首先讨论了系统的耗散性;接着分析了系统的平衡点并根据Routh-Hurwitz准则判断其局部稳定性;最后利用Lyapunov方法和Bendixson-Dulac判别法给出了平衡点的全局稳定性.     

一类离散的捕食与被捕食系统的稳定性与Neimark-Sacker分岔

本文利用映射的分岔理论讨论了一类离散的捕食与被捕食系统的动力学性质,分析了其正平衡点的稳定性,并讨论了Neimark-Sacker分岔的稳定性与方向。     

一类捕食者具有阶段结构的捕食——被捕食模型的全局性态分析

在假设捕食的受益是减少死亡下,建立了一类捕食种群具有阶段结构的捕食-被捕食模型,分析得到了不存在食饵种群情形下捕食者种群模型和食饵存在时捕食-被捕食模型的平衡点存在性和全局稳定性,并确定了决定模型动力学性态的捕食者种群基本再生数、捕食存在时的食饵种群净增长率以及食饵灭绝与否的捕食率阈值.     

一类捕食与被捕食系统的稳定性与分岔分析

本文利用Schur—Cohn—Jury引理及分岔理论讨论了一类捕食与被捕食系统的动力学性质，分析了其正平衡点的稳定性，并讨论了Neimark—Sacker分岔稳定性与方向。通过数值模拟验证了所得结果的正确性。     

带有疾病、分段常数变量的捕食-被捕食系统的稳定性和分支行为

本文研究一类带有疾病和分段常数变量的捕食-被捕食模型的稳定性和分支行为.首先通过计算得到捕食-被捕食模型对应的差分模型,利用线性稳定性理论讨论边界和正平衡点局部渐近稳定的充分条件.其次将食饵种群的出生率作为分支参数,使用分支理论研究差分模型在边界和正平衡点处产生鞍结点分支、翻转分支、Neimark-Sacker分支、Neimark-Sacker分支、鞍结点-Neimark-Sacker分支、鞍结点-翻转分支和翻转-Neimark-Sacker分支的充分条件.最后数值模拟验证理论分析的正确性,并展示模型复杂的动力学性态.     

捕食者与食饵都染病的捕食-被捕食模型分析

建立并分析了一个捕食者和食饵都染病的捕食-被捕食模型,求得了它的非负平衡点.利用Hurwitz判据,用特征根的方法得到了边界平衡点局部渐近稳定的充分条件.进一步利用LaSalle不变性原理获得了正平衡点全局渐近稳定的充分条件.     

具有扩散的捕食与被捕食系统的持续性和稳定性

本文研究了一类具有扩散和时滞的捕食与被捕食系统,证明了在适当条件下系统 是一致持续的,利用同伦技术证明了正平衡点的存在性,构造适当的Lyapunov函数获得 了正平衡点的局部和全局稳定的充分条件.     

具有恐惧的捕食与被捕食系统的动力学分析

建立一类考虑捕食者受到恐惧的捕食与被捕食模型,用于研究连续变化的恐惧对某一地区食物链的影响,其中恐惧主要影响捕食者的捕食率,并分析了模型的动力学性态,利用第二加性复合矩阵证明了正平衡点的全局稳定性.分析发现在失去大型食肉动物的食物链中,对中型捕食者加入恐惧,可以避免中型捕食者数量突然爆发,同时有利于食物链下方生物的多样性.     

Permanence of an SIR epidemic model with density dependent birth rate and distributed time delay

In this paper, we investigate the permanence of an SIR epidemic model with a density-dependent birth rate and a distributed time delay. We first consider the attractivity of the disease-free equilibrium and then show that for any time delay, the delayed SIR epidemic model is permanent if and only if an endemic equilibrium exists. Numerical examples are given to illustrate the theoretical analysis. The results obtained are also compared with those from the analog system with a discrete time delay.     

具有离散和分布时滞的Lotka-Volterra竞争系统

In this paper.the Lotka-Volterra competition system with discrete and distributed time delays is considered.By analyzing the characteristic equation of the linearized system,the local asymptotic stability of the positive equilibrium is investigated.Moreover,we discover the delays don't effect the stability of the equilibrium in the delay system.Finally,we can conclude that the positive equilibrium is global asymptotically stable in the delay system.     

Modelling and analysis of a prey–predator system with stage-structure and harvesting

In this paper, we have considered a prey–predator-type fishery model with Beddington–DeAngelis functional response and selective harvesting of predator species. We have established that when the time delay is zero, the interior equilibrium is globally asymptotically stable provided it is locally asymptotically stable. It is also shown that the time delay can cause a stable equilibrium to become unstable and even a switching of stabilities. Lastly, some numerical simulations are carried out.     

Mathematical Modeling and Analysis of an Epidemic Model with Quarantine, Latent and Media Coverage

Epidemic models are very important in today''s analysis of diseases. In this paper, we propose and analyze an epidemic model incorporating quarantine, latent, media coverage and time delay. We analyze the local stability of either the disease-free and endemic equilibrium in terms of the basic reproduction number $\mathcal{R}_{0}$ as a threshold parameter. We prove that if $\mathcal{R}_{0}<1,$ the time delay in media coverage can not affect the stability of the disease-free equilibrium and if $\mathcal{R}_{0}>1$, the model has at least one positive endemic equilibrium, the stability will be affected by the time delay and some conditions for Hopf bifurcation around infected equilibrium to occur are obtained by using the time delay as a bifurcation parameter. We illustrate our results by some numerical simulations such that we show that a proper application of quarantine plays a critical role in the clearance of the disease, and therefore a direct contact between people plays a critical role in the transmission of the disease.     

Analysis of a viral infection model with delayed immune response

It is well known that the immune response plays an important role in eliminating or controlling the disease after human body is infected by virus. In this paper, we investigate the dynamical behavior of a viral infection model with retarded immune response. The effect of time delay on stability of the equilibria of the system has been studied and sufficient condition for local asymptotic stability of the infected equilibrium and global asymptotic stability of the infection-free equilibrium and the immune-exhausted equilibrium are given. By numerical simulating,we observe that the stationary solution becomes unstable at some critical immune response time, while the delay time and birth rate of susceptible host cells increase, and we also discover the occurrence of stable periodic solutions and chaotic dynamical behavior. The results can be used to explain the complexity of the immune state of patients.     

Long term dynamics in a mathematical model of HIV-1 infection with delay in different variants of the basic drug therapy model

Infection with HIV-1, degrading the human immune system and recent advances of drug therapy to arrest HIV-1 infection, has generated considerable research interest in the area. Bonhoeffer et al. (1997) [1], introduced a population model representing long term dynamics of HIV infection in response to available drug therapies. We consider a similar type of approximate model incorporating time delay in the process of infection on the healthy T cells which, in turn, implies inclusion of a similar delay in the process of viral replication. The model is studied both analytically and numerically. We also include a similar delay in the killing rate of infected CD4⁺ T cells by Cytotoxic T-Lymphocyte (CTL) and in the stimulation of CTL and analyse two resulting models numerically.The models with no time delay present have two equilibria: one where there is no infection and a non-trivial equilibrium where the infection can persist. If there is no time delay then the non-trivial equilibrium is locally asymptotically stable. Both our analytical results (for the first model) and our numerical results (for all three models) indicate that introduction of a time delay can destabilize the non-trivial equilibrium. The numerical results indicate that such destabilization occurs at realistic time delays and that there is a threshold time delay beneath which the equilibrium with infection present is locally asymptotically stable and above which this equilibrium is unstable and exhibits oscillatory solutions of increasing amplitude.     

Non-selective harvesting in prey–predator models with delay

In this paper we are interested in studying the combined effects of harvesting and time delay on the dynamics of the generalized Gause type predator–prey models. It is shown that in these models the time delay may cause a stable equilibrium to become unstable and even a switching of stabilities, on the other hand harvesting effort has a stabilizing effect on the equilibrium if it is under the critical harvesting effort level. Simulations are carried out to explain some of the mathematical conclusions.     

Dynamical behavior of a delay virus dynamics model with CTL immune response

In this paper, the dynamical behavior of a virus dynamics model with CTL immune response and time delay is studied. Time delay is used to describe the time between the infected cell and the emission of viral particles on a cellular level. The effect of time delay on stability of the equilibria of the CTL immune response model has been studied and sufficient criteria for local asymptotic stability of the disease-free equilibrium, immune-free equilibrium and endemic equilibrium and global asymptotic stability of the disease-free equilibrium are given. Some conditions for Hopf bifurcation around immune-free equilibrium and endemic equilibrium to occur are also obtained by using the time delay as a bifurcation parameter. Numerical simulation with some hypothetical sets of data has been done to support the analytical findings.     

Bifurcation analysis of a modified Holling-Tanner predator-prey model with time delay

In this paper, a modified Holling-Tanner predator-prey model with time delay is considered. By regarding the delay as the bifurcation parameter, the local asymptotic stability of the positive equilibrium is investigated. Meanwhile, we find that the system can also undergo a Hopf bifurcation of nonconstant periodic solution at the positive equilibrium when the delay crosses through a sequence of critical values. In particular, we study the direction of Hopf bifurcation and the stability of bifurcated periodic solutions, an explicit algorithm is given by applying the normal form theory and the center manifold reduction for functional differential equations. Finally, numerical simulations supporting the theoretical analysis are also included.     

Ratio dependent predation :A bifurcation analysis

In this study, we have considered a prey-predator model reflecting the predator interference with discrete time delay. This delay is regarded as the lag due to gestation. In absence of delay, the criteria for existence of interior equilibrium and its global stability are derived. By choosing the delay as a bifurcation parameter, we have shown that a Hopf bifurcation may occur when the delay passes its critical value. Finally, we have derived the criteria for stability switches and verified the results through computer simulation.