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能源互联网呈现物理信息深度融合的趋势,为电力系统管理研究定义了新的研究框架。作为一个先进的复杂系统,能源互联网信息物理系统(Energy Internet Cyber-Physical System,ECPS)在其发展过程中面临着一些新的挑战,其中一个就是耦合结构下的风险管理。本文结合复杂网络理论和风险传递理论,着重在拓扑层面分析了ECPS跨空间交互机理,并在此基础上定义了交互路径和交互系数;接着建立了ECPS跨空间风险传递模型,量化描述了风险的传递和演化过程,并进行了风险影响评估;最后,通过仿真实验分析了三种不同交互系数节点故障的风险传递过程和不同攻击模式下系统的崩溃过程。对仿真结果的讨论阐述了能源互联网风险跨空间传递的特点,为更深入地研究ECPS的风险管理提供了参考。 相似文献
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A cDNA clone, p14--6, which has an antioncogene activity on the v--Ki--Ras oncogene-transformed malignant cell line DT, was found. This clone was recovered from the revertantR14 cells, which had been isolated by transfections of DT cells with a normal human fibro-blast cDNA library cloned in pcD2, an Okayama-Berg vector. When transfected into DT cells,p14--6 clone gave rise to phenotypical flat reversion in 5--15% of DT transfectant colo-nies. The p14--6--transfected flat cell line, RR, was proven to be a true revertant with signif-icantly reduced malignancy by in ritro and in riro malignancy tests. All other clones recov-ered from R14 cells were unable to cause this reversion. Molecular hybridizations showedthat the p14--6 was inserted into RR genome as tandem repeats, and no structural changewas found in the D--Ki--Ras oncogene in RR genome. These facts suggest that the antioncogeneactivity of the p14--6 clone on the DT cells may be exerted through expression of thecDNA contained in this clone. Possib 相似文献
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具有抗癌基因活性的一个cDNA克隆 总被引:2,自引:0,他引:2
本文发现了一个对受v-Ki-Ras基因恶性转化的小鼠成纤维细胞株DT具有抗癌基因活性的cDNA克隆p14-6。该cDNA克隆系从用正常人cDNA库转染DT造成的回复突变细胞株R14中回收得到的。用p14-6再次转染DT细胞株时,可使约5—15%的DT细胞发生形态回复突变。p14-6所造成的形态回复细胞系RR经体内和体外的恶性检验,证明其确为恶性程度较DT有显著降低的回复突变系。从R14中回收的其它cDNA克隆均不能产生此种回复突变。分子杂交显示p14-6以线形多聚体整合入RR基因组;v-Ki-Ras基因结构无可检出的变化。这些事实提示cDNA克隆p14-6对DT细胞的抗癌基因作用可能是由其cDNA的表达所引起。 对部分DT细胞被p14-6质粒转染后恶性不变的可能原因进行了讨论。 相似文献
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