改进了的捕食者-食饵系统的征税模型分析

探讨了Holling功能性反应的捕食者-食饵征税模型,修改了更合理的捕获函数.讨论了该系统生物经济平衡点的性态,正平衡点的局部渐近稳定性和全局渐近稳定性条件,并利用Pontrjagin最大值原理得到了最优税收策略.为可再生资源的合理开发利用提供了理论依据.     

一类带有阶段结构的扩散捕食系统的全局稳定性分析（英文）

本文研究一类带有反应扩散项、食饵具阶段结构的食饵捕食模型.通过运用Lyapunov直接法和构造合理的Lyapunov泛函,建立了系统边界平衡点和正平衡点的全局渐近稳定性,得到系统全局稳定的充分必要条件,提高已有的结论.     

一类具有Allee效应的捕食系统的稳定性分析及模拟

建立了食饵具有Allee效应的捕食模型,讨论了系统的有界性和平衡点的存在性.并证明了平衡点的局部渐近稳定性,进而通过构造Lyapunov函数分析了正平衡点的全局渐近稳定性,利用数值模拟讨论了Allee效应对系统的影响:Allee效应是系统的不稳定因素.     

疾病在食饵中流行的捕食与被捕食模型的分析

分析并建立了疾病在食饵中传播的生态-传染病模型,同时考虑到两种群都受密度制约因素的影响,讨论了模型解的有界性和各平衡点的存在性,利用Routh-Hurwitz判据证明了各平衡点的局部渐进稳定性,通过构造Lyapunov函数分析了各平衡点的全局渐进稳定性,得到了疾病存在与否的充分性条件.     

食饵具有避难所效应的生态-传染病模型的分析

建立并分析了疾病在食饵中传播、食饵考虑避难所效应的捕食与被捕食模型,捕食者不仅捕食感染食饵而且捕食易感食饵.讨论了系统的有界性和各平衡点的存在性,利用Routh-Hurwitz判据分析各平衡点的局部渐进稳定性,通过构造Lyapunov函数证明了各平衡点的全局渐进稳定性,并进行数值模拟以验证结论的正确性.     

具有时滞的生态-传染病模型的定性分析

针对一类疾病在食饵中传播而把食饵分为易感和染病的时滞生态-传染病模型,以时滞(即传染病在食饵种群中的潜伏期)作为分支参数,讨论了系统正平衡点在时滞τ=0时的局部渐近稳定性,在τ0时在一列临界值处发生了Hopf分支,并且对保持正平衡点稳定时时滞的范围也给出了估计.     

一类具有非线性传染率的时滞SIR模型的分析

分析并建立具有时滞及非线性传染率的SIR传染病模型.通过分析在无病平衡点和正平衡点处的特征方程,可得到在这两个平衡点处的局部渐近稳定性,然后我们得到了系统在两个平衡点处的全局渐近稳定性,最后我们证明了系统的持久性.     

一类基于比率的捕食-食饵系统的全局稳定性分析

研究一类基于比率和具第Ⅲ类功能性反应的捕食-食饵系统．通过分析正平衡点的局部稳定性给出了系统正平衡点全局渐近稳定以及系统存在极限环的条件．运用Hopf分支理论讨论了当正平衡点是非双曲型时的情形．     

具有时滞的生态-传染病模型的定性分析北大核心

针对一类疾病在食饵中传播而把食饵分为易感和染病的时滞生态-传染病模型,以时滞(即传染病在食饵种群中的潜伏期)作为分支参数,讨论了系统正平衡点在时滞τ=0时的局部渐近稳定性,在τ>0时在一列临界值处发生了Hopf分支,并且对保持正平衡点稳定时时滞的范围也给出了估计.     

具有食饵避难的Leslie-Gower捕食系统最优收获分析

研究了一类具有食饵避难的Leslie-Gower捕食与被捕食系统收获模型,利用Hurwitz判据,得到了正平衡点局部渐近稳定,进一步构造了适当的Lyapunov函数,证明了正平衡点的全局渐近稳定性.并且在捕获努力量假说下,对发生食饵避难的两种群同时捕获,考虑了生态经济平衡点的存在性和利用Pontryagin最大值原理对两种群进行最优收获,得到当贴现率为零时,既保持了生态平衡,又使得在渔业开发过程中取得最大经济利益.     

Cannibalism may control disease in predator population: result drawn from a model based study

In the present study, we propose and analyze a predator–prey system with disease in the predator population. To understand the role of cannibalism, we modify the model considering predator population is of cannibalistic type. Local and global stability around the biologically feasible equilibria are studied. The conditions for the persistence of the system are worked out. We also analyze and compare the community structure of the model systems with the help of ecological and disease basic reproduction numbers. Finally, through numerical simulation, we observe that inclusion of cannibalism in predator population may control the disease transmission in the susceptible predator population. Copyright © 2014 John Wiley & Sons, Ltd.     

A cannibalistic eco-epidemiological model with disease in predator population

In this present article, we propose and analyze a cannibalistic predator–prey model with disease in the predator population. We consider two important factors for the dynamics of predator population. The first one is governed through cannibalistic interaction, and the second one is governed through the disease in the predator population via cannibalism. The local stability analysis of the model system around the biologically feasible equilibria are investigated. We perform global dynamics of the model using Lyapunov functions. We analyze and compare the community structure of the system in terms of ecological and disease basic reproduction numbers. The existence of Hopf bifurcation around the interior steady state is investigated. We also derive the sufficient conditions for the permanence and impermanence of the system. The study reveals that the cannibalism acts as a self-regulatory mechanism and controls the disease transmission among the predators by stabilizing the predator–prey oscillations.     

一类具有Allee影响的捕食与被捕食模型

分析并建立了具有Allee影响的捕食与被捕食模型,被捕食者由于自身繁殖或是被捕食而具有了Allee效应,分别讨论了强Allee和弱Allee对被捕食种群的影响,讨论了解的有界性和各平衡点的存在性,并证明了各平衡点的局部渐近稳定性,进一步通过构造适当的Lyapunov函数分析了正平衡点E*的全局渐近稳定性.     

捕食者与食饵都染病的捕食-被捕食模型分析

建立并分析了一个捕食者和食饵都染病的捕食-被捕食模型,求得了它的非负平衡点.利用Hurwitz判据,用特征根的方法得到了边界平衡点局部渐近稳定的充分条件.进一步利用LaSalle不变性原理获得了正平衡点全局渐近稳定的充分条件.     

A predator–prey model with disease in the prey species only

A predator–prey model with transmissible disease in the prey species is proposed and analysed. The essential mathematical features are analysed with the help of equilibrium, local and global stability analyses and bifurcation theory. We find four possible equilibria. One is where the populations are extinct. Another is where the disease and predator populations are extinct and we find conditions for global stability of this. A third is where both types of prey exist but no predators. The fourth has all three types of individuals present and we find conditions for limit cycles to arise by Hopf bifurcation. Experimental data simulation and brief discussion conclude the paper. Copyright © 2006 John Wiley & Sons, Ltd.     

Dynamics of a predator–prey system with inducible defense and disease in the prey

Establishing and researching a population dynamical model based on the differential equation is of great significance. In this paper, a predator–prey system with inducible defense and disease in the prey is built from biological evolution and Eco-epidemiology. The effect of disease on population stability in the predator–prey system with inducible defense is studied. Firstly, we verify the positivity and uniform boundedness of the solutions of the system. Then the existence and stability of the equilibria are studied. There are no more than nine equilibrium points in the system. We use a sophisticated parameter transformation to study the properties of the coexistence equilibrium points of the system. A sufficient condition is established for the existence of Hopf bifurcation. Numerical simulations are performed to make analytical studies more complete.     

Disease‐selective predation may lead to prey extinction

In real world bio‐communities, predational choice plays a key role to the persistence of the prey population. Predator's ‘sense’ of choice for predation towards the infected and noninfected prey is an important factor for those bio‐communities. There are examples where the predator can distinguish the infected prey and avoids those at the time of predation. Based on the examples, we propose two mathematical models and observe the dynamics of the systems around biologically feasible equilibria. For disease‐selective predation model there is a high risk of prey extinction. On the other hand, for non‐disease selective predation both populations co‐exist. Local stability analysis and global stability analysis of the positive interior equilibrium are performed. Moreover, conditions for the permanence of the system are obtained. Finally, we conclude that strictly disease‐selective predation may not be acceptable for the persistence of the prey population. Copyright © 2005 John Wiley & Sons, Ltd.     

A modified Leslie–Gower predator–prey model with prey infection

The disease effect on ecological systems is an important issue from mathematical and experimental point of view. In this paper, we formulate and analyze a predator–prey model for the susceptible population, infected population and their predator population with modified Leslie–Gower (or Holling–Tanner) functional response. Mathematical analysis of the model equations with regard to invariance of nonnegativity and boundedness of solutions, local and global stability of the biological feasible equilibria and permanence of the system are presented. When the rate of infection crosses a critical value, we determine that the strictly positive interior equilibrium undergoes Hopf bifurcation. From our numerical simulations, we observe that the predation rate also plays an important role on the dynamic behavior of our system.