$QTAG$-Modules whose $h$-Pure-$S$-High Submodules Have Closure

A right module $M$ over an associative ring $R$ with unity is a $QTAG$-module if every finitely generated submodule of any homomorphic image of $M$ is a direct sum of uniserial modules. This article considers the closure of $h$-pure-$S$-high submodules of $QTAG$-modules. Here, we determine all submodules $S$ of a $QTAG$-module $M$ such that each closure of $h$-pure-$S$-high submodule of $M$ is $h$-pure-$\overline{S}$-high in $\overline{M}$. A few results of this theme give a comparison of some elementary properties of $h$-pure-$S$-high and $S$-high submodules.     

异常体液型冠心病患者细胞黏附分子含量的变化

根据维吾尔医学体液论,对305例维吾尔族冠心病患者进行异常体液分型分组,采用ELISA法,检测冠心病患者血浆可溶性细胞间黏附分子(slCAM-1)和可溶性血管细胞黏附分子(sVCAM-1)含量.观察不同异常体液型冠心病患者细胞黏附分子含量的变化.探讨细胞黏附分子在冠心病维吾尔医学异常体液分型中的临床意义.结果显示,305例维吾尔族冠心病患者中,异常黑胆质型冠心病70例、异常血液质型冠心病126例、异常胆液质型冠心病28例、异常黏液质型冠心病81例.与健康对照组比较.各组冠心病患者slCAM-1.sVCAM-1含量均有明显增加(P<0.01);在异常血液质组、异常胆液质组、异常黏液质组3组之间两两比较.均无显著性差异(p>0.05);但与其他3组进行两两比较.异常黑胆质组slCAM-1.sVCAM-1含量均高于其他各组(P<0.05).这表明.不同异常体液型冠心病患者细胞黏附分子含量均发生异常变化.异常黑胆质型冠心病患者细胞黏附分子含量的异常变化较其他异常体液型冠心病患者更为严重.     

Nucleon momentum distributions and elastic electron scattering form factors for some 1p-shell nuclei

The nucleon momentum distributions (NMD) and elastic electron scattering form factors of the ground state for 1p-shell nuclei with Z?=?N (such as ⁶Li, ¹⁰B, ¹²C and ¹⁴N nuclei) have been calculated in the framework of the coherent density fluctuation model (CDFM) and expressed in terms of the weight function $\left| {f( x )} \right|^2$ . The weight function has been expressed in terms of nucleon density distribution (NDD) of the nuclei and determined from the theory and the experiment. The feature of the long-tail behaviour at high-momentum region of the NMDs has been obtained by both the theoretical and experimental weight functions. The experimental form factors F(q) of all the considered nuclei are very well reproduced by the present calculations for all values of momentum transfer q. It is found that the contributions of the quadrupole form factors F _C2(q) in ¹⁰B and ¹⁴N nuclei, which are described by the undeformed p-shell model, are essential for obtaining a remarkable agreement between the theoretical and experimental form factors.     

新疆喀纳斯自然保护区盘星藻属植物新疆新记录

2012年8月,对于新疆喀纳斯国家级自然保护区,包括河流、湖泊和沼泽等地藻类植物进行了首次调查,共采集到50份藻类标本,并对其中的盘星藻属植物的形态特征进行了鉴定.结果发现,新疆盘星藻属植物新记录有2种6变种,它们分别为:短刺盘星藻短角变种(Pediastrum boryanumvar.brevicorne A.Braun),短刺盘星藻穿孔变种(Pediastrum boryanum var.perforatum Racib.),短刺盘星藻波缘变种(Pediastrum boryanumvar.undulatum Wille),具孔盘星藻[Pediastrum clathratum(Schroeter)Lemmermann],具孔盘星藻点纹变种(Pediastrum clathratum var.punctatumLemmermann),单角盘星藻[Pediastrum simplex(Meyen)Lemmermann],单角盘星藻具孔变种[Pediastrum simplexvar.duodenarium(Bailey)Rabenhorst],二角盘星藻网状变种(Pediastrum duplexvar.reticulatumLag.).这些新记录的发现,丰富了新疆盘星藻属植物的研究资料.     

AMIODARONE PHOTOTOXICITY TO HUMAN ERYTHROCYTES AND LYMPHOCYTES

Amiodarone (AD) therapy for cardiac arrhythmia frequently leads to cutaneous phototoxicity. Amiodarone and its metabolite, desethylamiodarone (DEA), photosensitized hemolysis of red blood cells (RBC) and were phototoxic to lymphocytes. Hemolysis photosensitized by 3.3 μ M AD was partially oxygen dependent and was partially quenched 5 m M sodium azide, 50 m M mannitol, superoxide dismutase (251 U/me e ) and catalase (1500 U/m e ), but was unaffected when H₂O was replaced by D₂O. These results suggest that membrane damage may be important in the in vivo phototoxicity to AD, that both oxygen dependent and oxygen independent mechanisms may operate, and that active oxygen species such as O₂ and hydrogen peroxide may be involved. Photohemolysis was more rapid in the presence of DEA than of AD. However, this may be due to the greater fragility of the cell membrane in the presence of DEA. The greater phototoxicity of DEA than AD towards lymphocytes was not due to greater membrane fragility.     

维药异常黑胆质成熟剂对异常黑胆质载体大鼠动物模型血液流变学指标的影响

 为了观察异常黑胆质成熟剂对异常黑胆质载体大鼠动物模型血液流变性及凝血功能的作用,选用健康Wistar大鼠,分为正常对照组、异常黑胆质模型组、药物干预组、自然恢复组,测定4组全血黏度、血浆黏度、全血还原黏度、红细胞聚集指数、变形指数、刚性指数、电泳指数、红细胞压积和血浆凝血酶原时间(PT),纤维蛋白原(FIB),活化部分凝血活酶时间(APTT)和血浆凝血酶时间(TT)等血液流变学指标并分析。结果显示,异常黑胆质成熟剂能有效控制异常黑胆质证大鼠模型血液黏度增高及凝血功能异常。     

普通物理实验的不确定度评定

本介绍了不确定度的有关概念,并结合分光计测棱镜折射率的例子,进行了具体分析,给出了测量不确定度的最终评定。     

原发性骨质疏松症患者维吾尔医异常体液病证诊断标准

 为建立原发性骨质疏松症维吾尔医异常体液病证量化诊断标准,以非疾病诊断临床相关信息为依据,对诊断明确的248例原发性骨质疏松症患者进行量化诊断入选项专家咨询研究。入选的证候要素进行多分类Logistic 回归分析、共线性诊断及主成分分析,使用主成分改进的多分类Logistic 回归分析方法确立各证候要素对证候的贡献度,根据优势比(OR 值)的大小分为原发性骨质疏松症各异常体液主症、次症;最后结合原发性骨质疏松“病”的自身特点,建立原发性骨质疏松症维医异常体液病证诊断标准。结果显示,原发性骨质疏松症临床上表现为腰背疼痛或周身骨骼疼痛,负荷增加时疼痛加重或活动受限,严重时翻身、起坐及行走有困难;可有身高缩短和驼背,脊柱畸形和伸展受限,低能量或非暴力骨折。其中,异常黑胆质型--主症:周身关节隐痛,面色晦暗,皮肤粗糙起鳞屑,多梦、噩梦,抑郁心态;次证:口味苦涩,舌质暗红,舌苔灰或黑色,大便干结,脉细、沉。异常血液质型--主症:腰背肢体胀痛,面色发红,皮肤较热,小便色橙黄,肢体沉重,脉象粗、有力;次症:睡眠尚少,口干,舌质暗红,舌苔少。异常黏液质型--主症:腰背肢体冷痛,肢体畏寒喜暖,面色暗淡,口黏,少气懒言;次证:面色晄白,舌体胖大边缘齿痕,睡眠较多,肢体麻木,脉象粗、沉、弱。异常胆液质型--主症:骨有灼痛、刺痛、疼有定处,皮肤较热,口干咽燥,口味甘苦,失眠,小便次数偏多,脉细、紧。次症:面色微黄,舌苔黄,盗汗。由此得出结论:通过量化诊断入选项专家咨询研究建立了原发性骨质疏松症的异常体液病证量化诊断标准。     

维药异常黑胆质成熟剂对乳腺增生模型大鼠乳腺组织病理形态及血清性激素水平的影响

 为探讨维药异常黑胆质成熟剂对乳腺增生模型大鼠的治疗作用及可能的作用机制,选用清洁级雌性Wistar 大鼠60 只,随机分为6 组：正常对照组、模型组、阳性对照组（三苯氧胺组）、异常黑胆质成熟剂高、中、低剂量组,采用大鼠右下肢肌肉注射雌、孕激素建立乳腺增生病模型,进行药物干预（三苯氧胺1.8 mg/kg,胆质成熟剂高、中、低剂量分别为3.75、7.5、15 g/kg）。运用光学显微镜观察维药异常黑胆质成熟剂对实验性乳腺增生症大鼠乳腺形态学的影响,放射免疫测定法检测其血清激素水平。结果表明,异常黑胆质成熟剂能减轻上皮细胞、间质纤维组织增生程度,改善模型大鼠乳腺组织病理形态学改变,调节血清激素水平,降低血清雌二醇（E₂）、黄体生成素（LH）、升高孕酮（P）、睾酮（T）、催乳素（PRL）及卵泡刺激素（FSH）水平,与模型组比较具有统计学差异（P<0.01 或P<0.05）。由此可见,异常黑胆质成熟剂对乳腺增生症大鼠的乳腺组织形态改变有一定的修复作用,同时能够降低大鼠血清雌激素水平,显著提高孕激素水平,且雌、孕激素比例失调状态也能够得到改善。