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General transport equations for a system of independent electrons in a random array of impurities of arbitrary range and in a magnetic field of arbitrary strength are derived on the basis of conventional perturbation theory, and the general structure of the transverse transport coefficients is discussed. Contrary to kinetic theory, within this framework it is easy to formulate “conserving” approximations, which include collision-broadening effects self-consistently. It is demonstrated, how for high magnetic field explicit results for the transverse transport coefficients can be obtained, which are free of the divergencies appearing in the kinetic theory.

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The archaeal exosome is formed by a hexameric RNase PH ring and three RNA binding subunits and has been shown to bind and degrade RNA in vitro. Despite extensive studies on the eukaryotic exosome and on the proteins interacting with this complex, little information is yet available on the identification and function of archaeal exosome regulatory factors.  相似文献   
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Ohne ZusammenfassungHerrn Prof. Dr.Klaus Wagner zum 60. Geburtstag gewidmet  相似文献   
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The experimental results of Dornhaus et al. on the dependence of the Auger recombination time on an applied magnetic field are explained within the frame of Takeshima's model by a proper inclusion of non-parabolicity and spin-splitting of the conduction band.  相似文献   
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The validity of assumptions involved in experimental evaluations of the g-factor of electrons in (100) surface inversion layers of p-silicon from Shubnikov-De Haas oscillations of the surface conductivity by the method of tilted megnetic fields is discussed on the basis of the transport theory of a two-dimensional electron-impurity system. The self-consistent Born approximation is shown to be insufficient to this task, and an alternative cumulant approach is discussed. Contrary to recent work, the cumulant approach is introduced in a simple manner, avoiding the path-integral formalism.  相似文献   
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We hypothesize that the energy strategy of a cell is a key factor for determining how, or if, the immune system interacts with that cell. Cells have a limited number of metabolic states, in part, depending on the type of fuels the cell consumes. Cellular fuels include glucose (carbohydrates), lipids (fats), and proteins. We propose that the cell's ability to switch to, and efficiently use, fat for fuel confers immune privilege. Additionally, because uncoupling proteins are involved in the fat burning process and reportedly in protection from free radicals, we hypothesize that uncoupling proteins play an important role in immune privilege. Thus, changes in metabolism (caused by oxidative stresses, fuel availability, age, hormones, radiation, or drugs) will dictate and initiate changes in immune recognition and in the nature of the immune response. This has profound implications for controlling the symptoms of autoimmune diseases, for preventing graft rejection, and for targeting tumor cells for destruction.  相似文献   
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