排序方式: 共有3条查询结果,搜索用时 0 毫秒
1
1.
Ch. A. Bell und F. L. Teed 《Fresenius' Journal of Analytical Chemistry》1882,21(1):127-129
Ohne Zusammenfassung 相似文献
2.
J. F. Sácher H. Lavers C. O. Bannister W. Mc Namara H. Koch Fr. Douglas Miles J. Waddell B. Oddo A. Beretta M. J. A. Müller Gr. Torossian W. N. Iwanow M. Siegfried W. Pozzi H. W. Woudstra J. M. Wilkie Teed A. V. Elsden J. F. Stansfield A. P. West J. A. Schaeffer H. Rubricius W. Stahl Fr. S. Schimerka P. Breteau P. Fleury v. Della Crose W. Elborne C. M. Warren S. Holzmann B. S. White und Carnelly 《Fresenius' Journal of Analytical Chemistry》1920,59(2-3):80-97
Ohne Zusammenfassung 相似文献
3.
Elisa?Fossale Pavlina?Wolf Janice?A?Espinola Tanya?Lubicz-Nawrocka Allison?M?Teed Hanlin?Gao Dorotea?Rigamonti Elena?Cattaneo Marcy?E?MacDonald Susan?L?CotmanEmail author 《BMC neuroscience》2004,5(1):57
Background
JNCL is a recessively inherited, childhood-onset neurodegenerative disease most-commonly caused by a ~1 kb CLN3 mutation. The resulting loss of battenin activity leads to deposition of mitochondrial ATP synthase, subunit c and a specific loss of CNS neurons. We previously generated Cln3 Δex7/8 knock-in mice, which replicate the common JNCL mutation, express mutant battenin and display JNCL-like pathology. 相似文献
1