A spectrum auction algorithm based on joint power control and beamforming in small cell networks

Spectrum auction is considered as a suitable approach to efficiently allocate spectrum among unlicensed users. However, in previous studies of spectrum auction, competition can hardly be reflected in the traditional spectrum allocation and the spectrum efficiency is still not high after the allocation. In this paper, we enhance the factor of competition in the auctions, in which bidders need to pay for the competition and the interference to macro cell users (MUs). We consider a communication system with one macro cell and several small cells, thus a licensed radio spectrum is shared by both MUs and small cell users (SUs). A truthful auction algorithm is proposed for spectrum allocation and the spectrum is divided into multiple channels in different time slots, so that SUs can make their choice for bidding under the guidance of history. In order to raise the communication quality, we propose a power control and beamforming algorithm in the heterogeneous network to enhance the system performance. Simulation results are presented to verify the effectiveness of the proposed algorithm in the small cell network.     

与Calderon—Zygmund型算子相关的Toeplitz型算子

设L为与Calder6n—Zygmund型相关的Toeplitz算子,通过建立Toeplitz算子的sharp极大函数的点态估计并应用该估计证明了当b∈BMO（Rn）时,Tb分别在加权Lp（w）空间,Morrey空间Lp,λ（P）和加权Morrey空间L（w）上有界．     

小度数点传递图的连通度

众所周知,ｋ（ｋ≤４）正则连通点传递图的连通度达到了它的正则度ｋ,本文证明了除Ｃｎ◎Ｋ２（ｎ≥４）外,每个５正则连通点传递图的连通度都是５,其中Ｃｎ◎Ｋ２是ｎ长圈与完全图Ｋ２的字典积。     

有界对称域上的BMO~p空间的乘子

该文得到在 Bergman度量下刻划有界 p-次平均振动函数 (BMOp)空间的乘子的几个等价条件 ,包含了文献 [3]中的 BMO2空间的结果     

A REMARK ON A BMO MARTINGALE

Let M = (Mt,Ft) be a uniformly integrable continuous martingale with MO = 0. For1 5 p < cot we setIIMllBMO. = '3p II[E[IMoo ~ MTIplFT]]'/Pll.,where the supremum is taken over all stopping times T.Set BMO. = {M: IIMllBMO. < co}. It is well known that BMO. = BMO, (VI S p 5 q).F'urthermore, all 11.llBMO. norms are equivalent andIIi ~~if;llMllBMO. = SUP T P(T < co)i'where the supremum is taken over all stopping times T satisfying P(T < co) > 0. In the laterwe shall simply …     

Small forcing creates neither strong nor Woodin cardinals

After small forcing, almost every strongness embedding is the lift of a strongness embedding in the ground model. Consequently, small forcing creates neither strong nor Woodin cardinals.

     

聚合物共混过程在线分析ARMA(p,q)模型的建立:小角光散射能量随时间变化的一维仿真研究

本文利用小角激光背散射 ( BSALS)在线分析系统对聚苯乙烯 ( PS)与顺丁橡胶( Pc BR)不相容体系共混过程中光散射强度 ( I( q)随时间的变化进行了分析 ,建立了自回归模型 ARMA( p,q) ,利用 AIC、BIC准则确定了模型的阶数 p和 q。用此模型对光散射强度进行模拟 ,经检验模拟结果与实验结果基本吻合。     

Gas Holdups of Small and Large Bubbles in a Large-scale Bubble Column with Elevated Pressure

Gas holdups of large bubbles and small bubbles were measured by means of dynamic gas disengagement approach in the pressured bubble column with a diameter of 0. 3 m and a height of 6.6 m. The effects of su-perficial gas velocity, liquid surface tension, liquid viscosity and system pressure on gas holdups of small bub-bles and large bubbles were investigated. The holdup of large bubbles increases and the holdup of small bub-bles decreases with an increase of liquid viscosity. Meanwhile, the holdup of large bubbles decreases with in-creasing the system pressure. A correlation for the holdup of small bubbles was obtained from the experimen-tal data.     

A Class of High-affinity Bicyclooctane G551D-CFTR Activators Identified by High Throughput Screening

The glycine-to-aspartic acid missense mutation at the codon 551(G551D) of the cystic fibrosis transmem-brane conductance regulator (CFTR) is one of the five most frequent cystic fibrosis (CF) mutations associated with a severe CF phenotype. To explore the feasibility of pharmacological correction of disrupted activation of CFTR chloride channel caused by G551D mutation, we developed a halide-sensitive fluorescence miniassay for G551D-CFTR in Fisher rat thyroid(FRT) epithelial cells for the discovery of novel activators of G551DCFTR. A class of bicyclooctane small molecule compounds that efficiently stimulate G551D-CFTR chloride channel activity was identified by high throughput screening via the FRT cell-based assay. This class of compounds selectively activates G551D-CFTR with a high affinity, whereas little effect of the compounds on wildtype CFTR can be seen. The discovery of a class of bicyclooctane G551D-CFTR activators will permit the analysis of structure-activity relationship of the compounds to identify ideal leads for in vivo therapeutic studies.