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In this paper, we analyze the nonlinear dynamic response of an orthotropic laminate in a simply supported boundary condition subject to thermal and mechanical loading. The equation of motion for the laminate’s deflection is obtained in a decoupled Duffing equation by means of a Galerkin-type method without Berger’s approximations. The Duffing equation incorporates an arbitrary thermal field, with both the in-plane and transverse temperature variations in a steady-state and a transient state. The formulation indicates that the transverse temperature variation produces an additional pressure load, while the in-plane temperature variation affects the system frequency. The equation allows for characterization of the laminate behaviors in nonlinear thermal buckling, thermal vibration and thermal mechanical response. 相似文献
63.
Gao Yuanwen 《Acta Mechanica Solida Sinica》2007,20(2):180-188
An analysis of buckling/snapping and bending behaviors of magneto-elastic-plastic interaction and coupling for cantilever rectangular soft ferromagnetic plates is presented.Based on the expression of magnetic force from the variational principle of ferromagnetic plates,the buckling and bending theory of thin plates,the Mises yield criterion and the increment theory for plastic deformation,we establish a numerical code to quantitatively simulate the behaviors of the nonlinearly multi-fields coupling problems by the finite element method.Along with the phenom- ena of buckling/snapping and bending,or the characteristic curve of deflection versus magnitude of applied magnetic fields being numerically displayed,the critical loads of buckling/snapping, and the influences of plastic deformation and the width of plate on these critical loads,the plastic regions expanding with the magnitude of applied magnetic field,as well as the evolvement of deflection configuration of the plate are numerically obtained in a case study. 相似文献
64.
弹性圆柱壳冲击扭转屈曲的突变模型 总被引:2,自引:0,他引:2
本文将突变理论用于处理冲击载荷下弹性结构的屈曲问题,文中建立了结构冲击屈曲的突变准则,认为临界阶跃载荷位于所提出突变系统的分叉集之中,并由此对两端固支的弹性圆柱壳扭转屈曲进行了分析,给出了不同初缺陷时的临界阶跃扭矩计算公式,通过对临界静态扭矩的计算,表明临界阶跃扭矩小于临界静态扭矩. 相似文献
65.
本文采用高阶剪切变形理论对正交各向异性中厚矩形板进行振动与稳定分析,数值计算采用样条有限点法,得出了六种不同边界条件矩形板的自振频率和屈曲载荷,并与相应的经典板理论的结果进行比较.结果说明横向剪切变形对复合材料层合板的影响与板的各向异性程度、板的宽厚比(b/h)、层合板的层数和板的支承条件有关,它随着层合板各向异性程度的增加而增加,随着层合板宽厚比的增加而逐渐消失. 相似文献
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通过对三种不同径厚比圆柱壳进行的流固冲击实验,分析了它们在流固冲击载荷下的动力响应特点,并与相应的高速撞击与静力屈曲实验作了简单的比较,分析了它们之间的异同点。同时文中对极值冲击倒塌特性进行了详细分析 相似文献
68.
《Journal of mass spectrometry : JMS》2017,52(7):ii-ii
Conformational change and modification of proteins are involved in many cellular functions. However, they can also have adverse effects that are implicated in numerous diseases. How structural change promotes disease is generally not well‐understood. This perspective illustrates how mass spectrometry (MS), followed by toxicological and epidemiological validation, can discover disease‐relevant structural changes and therapeutic strategies. We (with our collaborators) set out to characterize the structural and toxic consequences of disease‐associated mutations and post‐translational modifications (PTMs) of the cytosolic antioxidant protein Cu/Zn‐superoxide dismutase (SOD1). Previous genetic studies discovered >180 different mutations in the SOD1 gene that caused familial (inherited) amyotrophic lateral sclerosis (fALS). Using hydrogen–deuterium exchange with mass spectrometry, we determined that diverse disease‐associated SOD1 mutations cause a common structural defect – perturbation of the SOD1 electrostatic loop. X‐ray crystallographic studies had demonstrated that this leads to protein aggregation through a specific interaction between the electrostatic loop and an exposed beta‐barrel edge strand. Using epidemiology methods, we then determined that decreased SOD1 stability and increased protein aggregation are powerful risk factors for fALS progression, with a combined hazard ratio > 300 (for comparison, a lifetime of smoking is associated with a hazard ratio of ~15 for lung cancer). The resulting structural model of fALS etiology supported the hypothesis that some sporadic ALS (sALS, ~80% of ALS is not associated with a gene defect) could be caused by post‐translational protein modification of wild‐type SOD1. We developed immunocapture antibodies and high sensitivity top‐down MS methods and characterized PTMs of wild‐type SOD1 using human tissue samples. Using global hydrogen–deuterium exchange, X‐ray crystallography and neurotoxicology, we then characterized toxic and protective subsets of SOD1 PTMs. To cap this perspective, we present proof‐of‐concept that post‐translational modification can cause disease. We show that numerous mutations (N➔D; Q➔E), which result in the same chemical structure as the PTM deamidation, cause multiple diseases. Copyright © 2017 John Wiley & Sons, Ltd. 相似文献
69.
DDJTJQ是一个国内开发的组合结构静力分析和屈曲分析有限元程序系统,自1986年通过技术鉴定以来又有了许多发展。本文介绍该程序系统的功能和特点、求解能力以及在不同工程领域结构分析中的应用。 相似文献
70.