一类具有垂直传染与接种的DS—I—R传染病模型研究

本文研究了-类具有垂直传染与接种的疾病在多个易感群体中传播的DS-I-R传染病模型,得到了疾病流行的阈值.运用微分方程定性与稳定性理论分析了无病平衡点的局部稳定与全局渐近稳定性及存在唯一地方病平衡点与其全局渐近稳定性.     

具有免疫接种且总人口规模变化的SIR传染病模型的稳定性

讨论一类具有预防免疫接种且有效接触率依赖于总人口的SIR传染病模型,给出了决定疾病灭绝和持续生存的基本再生数σ的表达式,在一定条件下证明了疾病消除平衡点的全局稳定性,得到了唯一地方病平衡点的存在性和局部渐近稳定性条件.最后研究了具有双线性传染率和标准传染率的两个具体模型,并证明了当σ>1时该模型地方病平衡点的全局渐近稳定性.     

带有标准发生率和信息干预的时滞SIRS传染病模型的稳定性分析

本文考虑一类具有标准发生率和信息干预的时滞SIRS传染病模型.通过分析模型的特征方程,讨论无病平衡点和地方病平衡点局部渐近稳定性.应用Halanay不等式对无病平衡点的全局渐近稳定性进行证明.通过构造适当的Lyapunov函数讨论地方病平衡点全局渐近稳定性.最后通过数值模拟分析一些重要参数对疾病传播的影响.     

一类具有病毒变异的Logistic死亡率SEIR传染病模型研究分析

本文建立了一类具有病毒变异的Logistic死亡率SEIR传染病模型,借助Lyapunov函数和LaSalle''s不变原理,证明了无病平衡点全局稳定性.利用代数方法构造Lyapunov函数,证明了地方病平衡点全局稳定性.另外,通过数值模拟分析了参数对疾病传播的影响.     

疾病在食饵中流行的捕食与被捕食模型的分析

分析并建立了疾病在食饵中传播的生态-传染病模型,同时考虑到两种群都受密度制约因素的影响,讨论了模型解的有界性和各平衡点的存在性,利用Routh-Hurwitz判据证明了各平衡点的局部渐进稳定性,通过构造Lyapunov函数分析了各平衡点的全局渐进稳定性,得到了疾病存在与否的充分性条件.     

一类具有非线性传染率的SEIQR流行病模型的全局稳定性

研究了一类具有非线性传染率的SEIQR流行病数学模型,得到了疾病灭绝与否的基本再生数R_0的表达式,证明了无病平衡点和地方性平衡点的存在性及全局渐近稳定性.     

一类具有时滞的疾病感染的捕食-被捕食模型分析

研究了一类具有时滞的疾病感染的捕食-被捕食模型.首先讨论了系统的耗散性;接着分析了系统的平衡点并根据Routh-Hurwitz准则判断其局部稳定性;最后利用Lyapunov方法和Bendixson-Dulac判别法给出了平衡点的全局稳定性.     

一类具有双时滞的SEIRS传染病模型的分析

研究了一类具有双时滞的SEIRS传染病模型,利用对模型子系统的分析,得到了疾病灭绝与否的基本再生数,给出了无病平衡点的全局吸引性及地方病平衡点稳定性的存在条件,并证明了疾病的持久性.     

一种梅毒模型的稳定性分析

根据人类感染梅毒的方式建立了一种新的数学模型,整个人口被分成四个组:注射吸毒者,女性性工作者,性工作者的客人以及MSM人群.通过对模型的研究和分析得到了模型的基本再生数R_0,还进一步研究了平衡点的存在性和稳定性,当R_01时,无病平衡点是全局渐近稳定的,疾病将会被消除;当R_0 1时,疾病是一致持续的而且给出了地方病平衡点全局渐近稳定的充分性条件,疾病将持续流行.     

具有时滞和阶段结构的生态-流行病模型的稳定性及其Hopf分支

本文研究一个食饵具有阶段结构和捕食者染病的捕食者-食饵模型的稳定性,并讨论了由疾病的潜伏期引起的时滞对种群动力学性态的影响.通过分析特征方程,运用Hurwitz判定定理,讨论了该模型的平凡平衡点、捕食者灭绝平衡点、无病平衡点及地方病平衡点的局部稳定性,并得到了地方病平衡点附近Hopf分支存在的充分条件;通过构造适当的Lyapunov泛函,运用La Sall不变集原理,得到了这些平衡点全局稳定的充分条件.     

具有潜伏期的离散SEIR模型的稳定性

建立和研究了有年龄结构和潜伏期的离散SEIR模型,运用常差分线性方程组的理论,得到基本再生数R_0的表达式,证明了当R_0<1时,无病平衡点全局渐进稳定,当R_0>1时,无病平衡点不稳定,R_0>1且R_1<1时,地方病平衡点局部渐进稳定.     

一类带有潜伏期和部分免疫的传染病模型的全局分析

通过假设被接种者具有部分免疫,建立了一类具有潜伏期和接种的SEIR传染病模型,借助再生矩阵得到了确定此接种模型动力学行为的基本再生数.当基本再生数小于1时,模型只有无病平衡点;当基本再生数大于1时,除无病平衡点外,模型还有唯一的地方病平衡点.借助Liapunov函数,证明了无病平衡点和地方病平衡点的全局稳定性.     

两类带有确定潜伏期的SEIS传染病模型的分析

通过研究两类带有确定潜伏期的SEIS传染病模型,发现对种群的常数输入和指数输入会使疾病的传播过程产生本质的差异．对于带有常数输入的情形,找到了地方病平衡点存在及局部渐近稳定的阈值,证明了地方病平衡点存在时一定局部渐近稳定,并且疾病一致持续存在．对于带有指数输入的情形,发现地方病平衡点当潜伏期充分小时是局部渐近稳定的,当潜伏期充分大时是不稳定的．     

Analysis of a SIS epidemic model with stage structure and a delay

IntroductionAfter the pioneering work of Kermack-McKendrick on SIRS epidemiological models havebeen studied by many authorSll--31. There are some ldnds of disease which are only spread orhave more opportunities to be spread among children, for example, measles, chickenpox andscarlet fever, while others infectious diseases such as gonorrhea, swahilis are spread only amongadults. Consequently, realistic analysis of disease transmission in a population often requiresthe model to include stage …     

Mathematical analysis of an eco-epidemiological predator–prey model with stage-structure and latency

In this paper, an eco-epidemiological predator–prey model with stage structure for the prey and a time delay describing the latent period of the disease is investigated. By analyzing corresponding characteristic equations, the local stability of the trivial equilibrium, the predator-extinction equilibrium, the disease-free equilibrium and the endemic equilibrium is addressed. The existence of Hopf bifurcations at the endemic equilibrium is established. By using Lyapunov functionals and LaSalle’s invariance principle, sufficient conditions are obtained for the global asymptotic stability of the trivial equilibrium, the predator-extinction equilibrium, the disease-free equilibrium and the endemic equilibrium of the model.     

Stability analysis for an epidemic model with stage structure

An epidemic model with stage structure is formulated. The period of infection is partitioned into the early and later stages according to the developing process of infection, and the infectious individuals in the different stages have the different ability of transmitting disease. The constant recruitment rate and exponential natural death, as well as the disease-related death, are incorporated into the model. The basic reproduction number of this model is determined by the method of next generation matrix. The global stability of the disease-free equilibrium and the local stability of the endemic equilibrium are obtained; the global stability of the endemic equilibrium is got under the case that the infection is not fatal.     

Stochastic Epidemic SEIRS Models with a Constant Latency Period

In this paper, we consider the stability of a class of deterministic and stochastic SEIRS epidemic models with delay. Indeed, we assume that the transmission rate could be stochastic and the presence of a latency period of r consecutive days, where r is a fixed positive integer, in the “exposed” individuals class E. Studying the eigenvalues of the linearized system, we obtain conditions for the stability of the free disease equilibrium, in both the cases of the deterministic model with and without delay. In this latter case, we also get conditions for the stability of the coexistence equilibrium. In the stochastic case, we are able to derive a concentration result for the random fluctuations and then, using the Lyapunov method, to check that under suitable assumptions the free disease equilibrium is still stable.     

Global dynamics of a delayed SEIS infectious disease model with logistic growth and saturation incidence

In this paper, a delayed Susceptible‐Exposed‐Infectious‐Susceptible (SEIS) infectious disease model with logistic growth and saturation incidence is investigated, where the time delay describes the latent period of the disease. By analyzing corresponding characteristic equations, the local stability of a disease‐free equilibrium and an endemic equilibrium is discussed. The existence of Hopf bifurcations at the endemic equilibrium is established. By using the persistence theory for infinite dimensional dynamic systems, it is proved that if the basic reproduction number is greater than unity, the system is permanent. By means of suitable Lyapunov functionals and LaSalle's invariance principle, sufficient conditions are obtained for the global stability of the disease‐free equilibrium and the endemic equilibrium, respectively. Numerical simulations are carried out to illustrate the theoretical results. Copyright © 2015 John Wiley & Sons, Ltd.     

Global dynamics of a delayed eco‐epidemiological model with Holling type‐III functional response

In this paper, an eco‐epidemiological model with Holling type‐III functional response and a time delay representing the gestation period of the predators is investigated. In the model, it is assumed that the predator population suffers a transmissible disease. The disease basic reproduction number is obtained. By analyzing the corresponding characteristic equations, the local stability of each of feasible equilibria and the existence of Hopf bifurcations at the disease‐free equilibrium and the endemic‐coexistence equilibrium are established, respectively. By using the persistence theory on infinite dimensional systems, it is proved that if the disease basic reproduction number is greater than unity, the system is permanent. By means of Lyapunov functionals and LaSalle's invariance principle, sufficient conditions are obtained for the global stability of the endemic‐coexistence equilibrium, the disease‐free equilibrium and the predator‐extinction equilibrium of the system, respectively. Copyright © 2013 John Wiley & Sons, Ltd.     

Global stability of a delayed epidemic model with latent period and vaccination strategy

In this paper, a mathematical model describing the transmission dynamics of an infectious disease with an exposed (latent) period and waning vaccine-induced immunity is investigated. The basic reproduction number is found by applying the method of the next generation matrix. It is shown that the global dynamics of the model is completely determined by the basic reproduction number. By means of appropriate Lyapunov functionals and LaSalle’s invariance principle, it is proven that if the basic reproduction number is less than or equal to unity, the disease-free equilibrium is globally asymptotically stable and the disease fades out; and if the basic reproduction number is greater than unity, the endemic equilibrium is globally asymptotically stable and therefore the disease becomes endemic.