带有脉冲免疫和传染年龄的传染病模型的全局吸引性

讨论了带有脉冲免疫和传染年龄的传染病模型.传染类的恢复率是传染年龄的函数,当染病再生数小于1时,文章得到无病周期解是全局吸引的.如果总人口规模变化,也可得到类似的结论.最后,提出了带有脉冲免疫和传染年龄传染病模型待解决的问题.     

带有脉冲免疫和传染年龄的SIV传染病模型解的存在性

我们建立带有脉冲免疫和传染年龄的SIV传染病模型,这类传染病在潜伏期具有传染性,总人口规模依赖时间,并且传染类的康复率和传染年龄有关.我们证明此类模型解存在唯一.     

风险跨市场传染的综合测度、网络分析与趋势演化——基于商品期货的实证分析

在关联金融市场价格波动的相互溢出影响下，由风险传染而形成的多市场波动趋同现象正呈现多爆发、强影响、广扩散与久持续等系列特征，为了结合上述特征合理测度并有效应对这类风险跨市场传染现象，文章以我国商品期货为具体实证对象，分别从综合测度、网络分析与趋势演化三个角度对风险跨市场传染现象进行了系统研究。具体研究中，先立足Diebold-Yilmaz公式构建了风险跨市场传染测度指数，并结合网络分析、派系类分及趋势演化等方法，提出了风险跨市场传染现象的综合测度模型。在此基础上，以我国具有代表性的九个商品期货为对象，进行了新模型、新方法和新过程下的系列实证，并得出结论：商品期货风险跨市场传染现象具备网络性与时变性，并呈现市场差异性和影响不对称性；商品期货风险跨市场传染现象派系类分具备总体一致性与个体交叉性；商品期货风险跨市场传染主要通过点传染及派系传染进行。     

带有免疫和传染年龄的传染病模型

建立了带有免疫和传染年龄的传染病模型,这种传染病带有病原体Ⅰ或Ⅱ,病原体Ⅰ可发展为病原体Ⅱ,得到了无病平衡态全局稳定和局部稳定的条件.当病原体Ⅰ不发展为病原体Ⅱ时,得到了病原体Ⅰ类平衡态的稳定性依赖于病原体Ⅱ类的基本再生指数.     

具有常数输入的SEIR模型的稳定性分析

讨论了易感者类和潜伏者类均为常数输入,潜伏期、染病期和恢复期均具有传染力,且传染率为一般传染率的SEIR传染病模型.利用Hurwitz判据证明了地方病平衡点的局部渐近稳定性,进一步利用复合矩阵理论得到了地方病平衡点全局渐近稳定的充分条件.     

供应链上两类不同信用风险的关联及传染效应研究

在一个典型的存在资金约束的二级供应链上,通常存在两类不同的信用风险:一是供应商向银行信用贷款所形成的信贷信用风险;二是供应商为零售商提供商业信用而形成的商业信用风险。本文针对上述两类不同信用风险之间的关联性及传染问题展开研究,揭示了两类风险之间的传染机理,度量了风险的传染强度,并分析了传染强度的影响因素。研究表明:两类不同信用风险之间的关联性,导致供应链上存在由非同类信用风险构成的关联信用风险(Different types of associated credit risk),本文简称为供应链上的D类关联信用风险。结合数值分析发现:D类关联信用风险的传染强度与供应商的生产成本及无风险利率正相关,而与商品的市场价格负相关。特别地,当市场需求服从指数分布时,关联信用风险的传染强度与商业信用成本和零售商的特质有关,供应商通过调整商业信用成本和选择零售商类型可以降低D类关联信用风险的传染强度,进而降低自身的银行信贷风险。本文将同类型信用风险之间的关联和传染问题引申到不同类型的信用风险之间,进一步深化和拓展了关联信用风险的内涵。     

PFS聚类法在医疗管理中的应用

ＰＦＳ聚类法在医疗管理中的应用赵文霞，吕蓬（华北电力学院）本文以能反映医疗水平的四项指标为基础，采用聚类法，对保定市十二个医疗科（肿瘤、传染、呼吸等科）的医疗情况进行了聚类分析，并通过Ｐｕｇ值确定了最佳分类数。分类结果与客观实际相符。在聚类分析中，所...     

基于银行-资产二分网络的银行间风险传染研究

银行之间越来越紧密的联系形成了复杂的网络,使得风险能够在网络中迅速传染,造成大规模的级联失效.目前已有的大多数研究都基于银行间资产负债表关联来分析银行间风险传染,而银行间持有相同资产的关联作为一种重要的银行间风险传染的渠道,往往没有引起足够的重视.基于持有相同资产关联,建立银行-资产二分网络研究银行间风险传染的机理特征.首先设计了风险传染模型,接着收集了244家主要银行2016年的资产价值数据作为模型仿真的基础.仿真结果表明银行间持有相同资产关联会造成银行间风险传染,继而引发大规模的银行级联破产,其危害程度和传染速度随着冲击大小和溢出效应强度的增加而增加.同时,按照资产占比将244家银行聚成5类并分析各类银行的风险特点.分析对监管部门进行合理分配监管资源具有一定的参考意义.     

一类含有非线性传染率的传染病模型的全局稳定性

讨论了一类带有非线性传染率的SIRS型传染病模型,得到了无病平衡点和地方病平衡点存在的阈值条件,借助构造Dulac函数和Liapunov函数,找到了两类平衡点全局渐近稳定的充要条件.     

金融风险在股票市场的传染效应及联动行为分析

近年来金融危机频发并表现出了易传染性,引起了众多学者的高度关注。以动态条件相关模型研究美欧股市与中、日、韩股市间的时变相关性,并结合内生多重结构突变模型划分危机传染阶段,选用溢出指数模型分析股市间的风险溢出特性;随后,定义股市间相互影响的联动模式并构建不同传染阶段的加权有向网络图分析股市间的联动行为。研究表明:美欧股市对中日韩股市有明显的传染效应,被传染的速度和持续时间均不相同;金融传染和风险溢出展现出一定的不一致性,危机期间日股的风险溢出效应强于美股;传染效应在联动网络中表现为联动模式的高聚类性和高联动性,相比欧债危机,次贷危机时期股市间展现出更强的联动行为;日股与美欧股市在两次危机中均表现出最强的联动性,其所受影响也最大。     

Dynamics of an age‐structured host‐vector model for malaria transmission

In this paper, we propose a host‐vector model for malaria transmission by incorporating infection age in the infected host population and nonlinear incidence for transmission from infectious vectors to susceptible hosts. One novelty of the model is that the recovered hosts only have temporary immunity and another is that successfully recovered infected hosts may become susceptible immediately. Firstly, the existence and local stability of equilibria is studied. Secondly, rigorous mathematical analyses on technical materials and necessary arguments, including asymptotic smoothness and uniform persistence of the system, are given. Thirdly, by applying the fluctuation lemma and the approach of Lyapunov functionals, the threshold dynamics of the model for a special case were established. Roughly speaking, the disease‐free equilibrium is globally asymptotically stable when the basic reproduction number is less than one and otherwise the endemic equilibrium is globally asymptotically stable when no reinfection occurs. It is shown that the infection age and nonlinear incidence not only impact on the basic reproduction number but also could affect the values of the endemic steady state. Numerical simulations were performed to support the theoretical results.     

An Age-structured Mathematical Model for the Within Host Dynamics of Malaria and the Immune System

In the paper, we use a mathematical model to study the population dyna mics of replicating malaria parasites and their interaction with the immune cells within a human host. The model is formulated as a system of age-structured partial differential equations that are then integrated over age to obtain a system of nonlinear delay differential equations. Our model incorporates an intracellular time delay between the infection of the red blood cells by the merozoites that grow and replicate within the infected cells to produce new merozoites. The infected red blood cells burst approximately every 48 h releasing daughter parasites to renew the cycle. The dynamical processes of the parasites within the human host are subjected to pressures exerted by the human immunological responses. The system is then solved using a first-order, finite difference method to give a discrete system. Numerical simulations carried out to illustrate stability of the system reveal that the populations undergo damped oscillations that stabilise to steady states.      

一类带有感染年龄SIR模型最优化免疫策略的存在性

对于一个免疫策略来讲,付出(单位时间内接种疫苗的数量)和效果(再生数的大小)是两个重要概念.在给定的费用下找到带有最小再生数的策略和在给定的再生数下找到最小费用的策略是两个最优问题.对一个确定的免疫策略来说,人群中的易感群体和染病群体会趋于相对稳定的状态.当一种疾病侵袭已免疫人群时,用带有感染年龄的SIR模型去描述这类疾病的传播更为准确.因此,本文研究了一类带有感染年龄的SIR模型,得到了最优化策略的存在性.     

Threshold behaviour of a SI epidemiological model with two structuring variables

In this article, we study a SI epidemic model describing the spread of a disease in a perfectly mixed managed population, representing an animal herd in a fattening farm. The epidemic process is characterized by a non-neglectable and variable incubation period, during which individuals are infectious but cannot be easily detected. The susceptible and infected populations are structured according to age and, for infected, to time remaining before the end of the incubation, where they show detectable clinical signs. We study the well posedness and the asymptotic behaviour of the problem and show that in some cases, even if the farm is fed with healthy animals, disease persistence can occur. We give an explicit formula for the basic reproduction number \({\mathcal{R}_0}\) and the biological interpretation of this threshold on a specific example. We finally illustrate the asymptotic behaviour of the model by numerical simulations.     

A nonlinear AIDS epidemic model with screening and time delay

A nonlinear mathematical model to study the effect of time delay in the recruitment of infected persons on the transmission dynamics of HIV/AIDS is proposed and analyzed. In modeling the dynamics, the population is divided into four subclasses: the susceptibles, the HIV positives or infectives that do not know they are infected, the HIV positives that know they are infected and the AIDS patients. Susceptibles are assumed to become infected via sexual contacts with (both types of) infectives. The model is analyzed using stability theory of delay differential equations. Both the disease-free and the endemic equilibria are found and their stability is investigated. It is shown that the introduction of time delay in the model has a destabilizing effect on the system and periodic solutions can arise by Hopf bifurcation. Numerical simulations are also carried out to investigate the influence of key parameters on the spread of the disease, to support the analytical conclusion and to illustrate possible behavioral scenario of the model.     

自适应网络上的SIV模型动力学

建立了自适应网络上的SIV （susceptible-infected-vaccinated）模型.在该模型中,接种的个体由于疫苗的不完全有效而会被感染.假设只有易感态个体采用重连机制进行自我保护,这一机制使得网络拓扑结构和疾病传播共同演化.通过平均场近似并求解方程,发现了丰富的动力学现象,如双稳态和周期振荡.网络的演化也趋于单峰度分布和负相关性.     

Vaccination strategies based on feedback control techniques for a general SEIR-epidemic model

This paper presents several simple linear vaccination-based control strategies for a SEIR (susceptible plus infected plus infectious plus removed populations) propagation disease model. The model takes into account the total population amounts as a refrain for the illness transmission since its increase makes more difficult contacts among susceptible and infected. The vaccination control objective is the asymptotically tracking of the removed-by-immunity population to the total population while achieving simultaneously that the remaining populations (i.e. susceptible plus infected plus infectious) tend asymptotically to zero.     

On vaccination controls for the SEIR epidemic model

This paper presents a simple continuous-time linear vaccination-based control strategy for a SEIR (susceptible plus infected plus infectious plus removed populations) disease propagation model. The model takes into account the total population amounts as a refrain for the illness transmission since its increase makes more difficult contacts among susceptible and infected. The control objective is the asymptotically tracking of the removed-by-immunity population to the total population while achieving simultaneously the remaining population (i.e. susceptible plus infected plus infectious) to asymptotically tend to zero.     

Effect of emergent carrying capacity in an eco‐epidemiological system

The paper explores an eco‐epidemiological model of a predator–prey type, where the prey population is subject to infection. The model is basically a combination of S‐I type model and a Rosenzweig–MacArthur predator–prey model. The novelty of this contribution is to consider different competition coefficients within the prey population, which leads to the emergent carrying capacity. We explicitly separate the competition between non‐infected and infected individuals. This emergent carrying capacity is markedly different to the explicit carrying capacities that have been considered in many eco‐epidemiological models. We observed that different intra‐class and inter‐class competition can facilitate the coexistence of susceptible prey‐infected prey–predator, which is impossible for the case of the explicit carrying capacity model. We also show that these findings are closely associated with bi‐stability. The present system undergoes bi‐stability in two different scenarios: (a) bi‐stability between the planner equilibria where susceptible prey co‐exists with predator or infected prey and (b) bi‐stability between co‐existence equilibrium and the planner equilibrium where susceptible prey coexists with infected prey; have been discussed. The conditions for which the system is to be permanent and the global stability of the system around disease‐free equilibrium are worked out. Copyright © 2015 John Wiley & Sons, Ltd.