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1.
In this study, we investigate a pine wilt transmission model with nonlinear incidence rates. The stability of the system is analyzed for disease-free and endemic equilibria. It is proved that the global dynamics are completely by the basic reproduction number R0R0. If R0R0 is less than one, the disease-free equilibrium is globally asymptotically stable, and in such a case, the endemic equilibrium does not exist. If R0R0 is greater than one, the disease persists and the unique endemic equilibrium is globally asymptotically stable.  相似文献   

2.
In this paper, applying two types of Lyapunov functional techniques to an SIRS epidemic model with graded cure and incomplete recovery rates, we establish complete global dynamics of the model whose threshold parameter is the basic reproduction number R0R0 such that the disease-free equilibrium is globally asymptotically stable when R0?1R0?1, and the endemic equilibrium is globally asymptotically stable when R0>1R0>1.  相似文献   

3.
A model with acute and chronic stages in a population with exponentially varying size is proposed. An equivalent system is obtained, which has two equilibriums: a disease-free equilibrium and an endemic equilibrium. The stability of these two equilibriums is controlled by the basic reproduction number R0R0. When R0<1R0<1, the disease-free equilibrium is globally stable. When R0>1R0>1, the disease-free equilibrium is unstable and the unique endemic equilibrium is locally stable. When R0>1R0>1 and γ=0,α=0γ=0,α=0, the endemic equilibrium is globally stable in Γ0Γ0.  相似文献   

4.
In this paper, we investigate the dynamical behavior of a virus infection model with delayed humoral immunity. By using suitable Lyapunov functional and the LaSalle?s invariance principle, we establish the global stabilities of the two boundary equilibria. If R0<1R0<1, the uninfected equilibrium E0E0 is globally asymptotically stable; if R1<1<R0R1<1<R0, the infected equilibrium without immunity E1E1 is globally asymptotically stable. When R1>1R1>1, we obtain the sufficient conditions to the local stability of the infected equilibrium with immunity E2E2. The time delay can change the stability of E2E2 and lead to the existence of Hopf bifurcations. The stabilities of bifurcating periodic solutions is also studied. We check our theorems with numerical simulations in the end.  相似文献   

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In this paper, a mathematical model for HILV-I infection of CD4+ T-cells is investigated. The force of infection is assumed be of a function in general form, and the resulting incidence term contains, as special cases, the bilinear and the saturation incidences. The model can be seen as an extension of the model [Wang et al. Mathematical analysis of the global dynamics of a model for HTLV-I infection and ATL progression, Math. Biosci. 179 (2002) 207-217; Song, Li, Global stability and periodic solution of a model for HTLV-I infection and ATL progression, Appl. Math. Comput. 180(1) (2006) 401-410]. Mathematical analysis establishes that the global dynamics of T-cells infection is completely determined by a basic reproduction number R0R0. If R0?1R0?1, the infection-free equilibrium is globally stable; if R0>1R0>1, the unique infected equilibrium is globally stable in the interior of the feasible region.  相似文献   

6.
A virus dynamics model with Beddington–DeAngelis functional response and delays is introduced. By analyzing the characteristic equations, the local stability of an infection-free equilibrium and a chronic-infection equilibrium of the model is established. By using suitable Lyapunov functionals and the LaSalle invariance principle, we show that the infection-free equilibrium is globally asymptotically stable if R0?1R0?1 and the chronic-infection equilibrium is globally asymptotically stable if R0>1R0>1. Numerical simulations are also given to explain our results.  相似文献   

7.
In this study, we consider the population dynamics of an invasive species and a resident species, which are modeled as a diffusive competition process in a radially symmetric setting with a free boundary. We assume that the resident species undergoes diffusion and growth in RnRn, while the invasive species initially exists in a finite ball, but invades the environment with a spreading front evolving according to a free boundary. When the invasive species is inferior, we show that if the resident species is already well established initially, then the invader can never invade deep into the underlying habitat, thus it dies out before its invading front reaches a certain finite limiting position. When the invasive species is superior, a spreading–vanishing dichotomy holds, and sharp criteria for spreading and vanishing with d1d1, μ  , and u0u0 as variable factors are obtained, where d1d1, μ  , and u0u0 are the dispersal rate, expansion capacity, and initial number of invaders, respectively. In particular, we obtain some rough estimates of the asymptotic spreading speed when spreading occurs.  相似文献   

8.
In this paper, we propose a novel time delayed HIV/AIDS mathematical model and further analyze the effect of vaccination and ART (antiretroviral therapy) on this time delayed model, in which the time delay is due to the strong immune response to AIDS for the HIV-infected-aware because of the good physical conditions. We introduce the different stages of the period of AIDS infection having different abilities of transmitting disease, which reflects the developing progress of AIDS infection more realistically. By using suitable Lyapunov functionals and the LaSalle invariant principle, we obtain the basic reproduction number R0R0 and derive that if R0<1R0<1 and some parameters satisfy a given condition, the disease-free equilibrium is globally asymptotically stable, while the disease will be died out. Numerical simulations are carried out to verify the obtained stability criteria and demonstrate the effect of the vaccination rate and R0R0 and the ART on the infective individuals.  相似文献   

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10.
Let H:=H0+VH:=H0+V and H:=H0,+VH:=H0,+V be respectively perturbations of the unperturbed Schrödinger operators H0H0 on L2(R3)L2(R3) and H0,H0, on L2(R2)L2(R2) with constant magnetic field of strength b>0b>0, and V a complex relatively compact perturbation. We prove Lieb–Thirring type inequalities on the discrete spectrum of H   and HH. In particular, these estimates give a priori information on the distribution of eigenvalues around the Landau levels of the operator, and describe how fast sequences of eigenvalues converge.  相似文献   

11.
Protein translocation in cells has been modelled by Brownian ratchets  . In such models, the protein diffuses through a nanopore. On one side of the pore, ratcheting molecules bind to the protein and hinder it to diffuse out of the pore. We study a Brownian ratchet by means of a reflected Brownian motion (Xt)t0(Xt)t0 with a changing reflection point (Rt)t0(Rt)t0. The rate of change of RtRt is γ(XtRt)γ(XtRt) and the new reflection boundary is distributed uniformly between RtRt and XtXt. The asymptotic speed of the ratchet scales with γ1/3γ1/3 and the asymptotic variance is independent of γγ.  相似文献   

12.
This paper is concerned with the Cauchy problem for the fast diffusion equation ut−Δum=αup1utΔum=αup1 in RNRN (N≥1N1), where m∈(0,1)m(0,1), p1>1p1>1 and α>0α>0. The initial condition u0u0 is assumed to be continuous, nonnegative and bounded. Using a technique of subsolutions, we set up sufficient conditions on the initial value u0u0 so that u(t,x)u(t,x) blows up in finite time, and we show how to get estimates on the profile of u(t,x)u(t,x) for small enough values of t>0t>0.  相似文献   

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In this paper, a SEIV epidemic model with a nonlinear incidence rate is investigated. The model exhibits two equilibria, namely, the disease-free equilibrium and the endemic equilibrium. It is shown that if the basic reproduction number R0<1R0<1, the disease-free equilibrium is globally asymptotically stable and in such a case the endemic equilibrium does not exist. Moreover, we show that if the basic reproduction number R0>1R0>1, the disease is uniformly persistent and the unique endemic equilibrium of the system with saturation incidence is globally asymptotically stable under certain conditions.  相似文献   

18.
In this paper we prove that given any two point lattices Λ1⊂RnΛ1Rn and Λ2⊂Rn−kΛ2Rnk, there is a set of k   vectors {v1,…,vk}⊂Λ1{v1,,vk}Λ1 such that Λ2Λ2 is, up to similarity, arbitrarily close to the projection of Λ1Λ1 onto the orthogonal complement of the subspace spanned by {v1,…,vk}{v1,,vk}. This result extends the main theorem of Sloane et al. (2011) [1] and has applications in communication theory.  相似文献   

19.
Let kk be a field of characteristic zero and RR a factorial affine kk-domain. Let BB be an affineRR-domain. In terms of locally nilpotent derivations, we give criteria for BB to be RR-isomorphic to the residue ring of a polynomial ring R[X1,X2,Y]R[X1,X2,Y] over RR by the ideal (X1X2−φ(Y))(X1X2φ(Y)) for φ(Y)∈R[Y]?Rφ(Y)R[Y]?R.  相似文献   

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