电子在蛋白质分子中的迁移机理及其特性

ATP水解作用所释放的能量能引起蛋白质分子中额外电子的激发,其激发与氨基酸残基晶格畸变相互作用,使额外电子自陷成孤子,这孤子在附着有供体(D)和受体(A)的蛋白质分子中运动时,由于孤子与供体电子的相互作用而"自局域"在孤子上进行迁移,从而可把电子供给受体.使用作者提出的孤子理论研究了这种迁移的特征,计算了在这种非平衡态过程中由孤子迁移电子的速率和动能系数,得到了一些有趣的结果,揭示了这种迁移的本质,求得了迁移的大小.

Mechanism and properties of electron transfer in protein molecules

Excitation of extra-electron is caused by the energy released in ATP hydrolysis in protein molecules. The excitation can self-trap as a soliton through interaction with deformation of amino acids. When donors and acceptors are distributed along the protein molecular chains close enough, the electrons in the donors can be transferred by the soliton due to the interaction between them. Authors calculate the speed and kinetic coefficient of the electron transfer from the donors and acceptors in the molecular chains with cubic anharmonicity by non-equilibrium statistical theory. Some interesting results are obtained.