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On the Global Stability of Cooperative Systems   总被引:7,自引:0,他引:7  
Suppose that F is a C1 cooperative vector field on X, whereX = Rn or Int , or [[p, q]].We prove that the unique equilibrium of F is globally asymptoticallystable if and only if every forward semi-orbit has compact closurein X.  相似文献   
2.
Summary For multidimensional second-order differential equations we present new conditions for the boundedness and convergence of all solutions and their derivatives, and the existence of periodic solutions. In particular, for scalar case we give the necessary and sufficient conditions for the boundedness and convergence of all solutions and their derivatives. These results are used to improve a result of Levin and Nohel for the reactor dynamics and a result of Holmes for the evolution equations derived from a conveying fluid and to give an answer to the conjecture on the continuous stirred tank proposed by Uppal etc.  相似文献   
3.
梅继法  黎军顽  倪玉山  王华滔 《物理学报》2011,60(6):66104-066104
基于嵌入原子势考察体心立方(bcc)金属Ta的广义层错能和广义孪晶能并获得广义层错能和广义孪晶能曲线. 研究表明,bcc Ta的广义层错能曲线与面心立方金属的广义层错能曲线有明显差异,Ta的广义层错能曲线不存在明显的能量极小值,位错主要以全位错的形式发射. 不同原子厚度的广义孪晶能曲线表明4个原子层的孪晶能曲线开始出现亚稳定的能量极小值,5个原子层的孪晶能曲线出现稳定的能量极小值. 为进一步验证广义层错能和广义孪晶能曲线揭示的塑性变形机理,采用准连续介质力学多尺度方法研究Ⅱ型裂纹尖端的初始塑性变形过程. 关键词: 广义层错能 广义孪晶能 体心立方金属钽 Ⅱ型裂纹  相似文献   
4.
High density boron carbide nanowires are grown by an improved carbon thermal reduction technique. Transmission electron microscopy and electron energy lose spectroscopy of the sample show that the synthesized nanowires are B4 C with good crystallization. The field emission measurement for an individual boron nanowire is performed by using a Pt tip installed in the focused ion beam system. A field emission current with enhancement factor of 10^6 is observed and the evolution process during emission is also carefully studied. Furthermore, a two-step field emission with stable emission current density is found from the high-density nanowire film. Our results together suggest that boron carbide nanowires are promising candidates for electron emission nanodevices.  相似文献   
5.
陆怀宝  黎军顽  倪玉山  梅继法  王洪生 《物理学报》2011,60(10):106101-106101
本文采用多尺度准连续介质法(quasi-continuum method, QC)模拟体心立方(body-centered-cubic, bcc)金属钽(Ta)Ⅱ型裂纹尖端位错的形核与发射过程,获得位错发射位置与应力强度因子关系曲线,分析裂纹尖端缺陷萌生过程,研究全位错分解以及扩展位错形成机理. 位错活动在不同阶段表现出不一致的特征,新位错的发射对于位错运动具有促进作用. 研究表明,裂纹扩展初始阶段首先萌生点缺陷,点缺陷随着加载强度增加会萌生新的点缺陷,点缺陷最终运动到边界,导致Ⅱ型断裂破坏. 在全位错发射之前有不全位错的形核与发射表明全位错的分解分步进行,从势能曲线上来看,也就是两个极小值点的形成机理不同. 关键词: 多尺度 准连续介质法 Ⅱ型裂纹 扩展位错  相似文献   
6.
This paper studies the asymptotic behaviour of an analytic order-preservingdiscrete-time dynamical system in Rn, which is usually generatedby a periodic cooperative system. The author proves that forsuch a dynamical system, if every fixed point is Liapunov stableand every positive semi-orbit has compact closure, then everypositive semi-orbit converges. This result does not requirethe assumption ‘strongly’ and gives an affirmativeanswer to the conjecture proposed by the author in [17] forthe analytic case.  相似文献   
7.
Dopamine D1 receptor (D1DR) and D2 receptor (D2DR) are closely associated with pain modulation, but their exact effects on neuropathic pain and the underlying mechanisms remain to be identified. Our research revealed that intrathecal administration of D1DR and D2DR antagonists inhibited D1–D2DR complex formation and ameliorated mechanical and thermal hypersensitivity in chronic constriction injury (CCI) rats. The D1–D2DR complex was formed in the rat spinal cord, and the antinociceptive effects of D1DR and D2DR antagonists could be reversed by D1DR, D2DR, and D1–D2DR agonists. Gαq, PLC, and IP3 inhibitors also alleviated CCI-induced neuropathic pain. D1DR, D2DR, and D1–D2DR complex agonists all increased the intracellular calcium concentration in primary cultured spinal neurons, and this increase could be reversed by D1DR, D2DR antagonists and Gαq, IP3, PLC inhibitors. D1DR and D2DR antagonists significantly reduced the expression of p-PKC γ, p-CaMKII, p-CREB, and p-MAPKs. Levo-corydalmine (l-CDL), a monomeric compound in Corydalis yanhusuo W.T. Wang, was found to obviously suppress the formation of the spinal D1–D2DR complex to alleviate neuropathic pain in CCI rats and to decrease the intracellular calcium concentration in spinal neurons. l-CDL-induced inhibition of p-PKC γ, p-MAPKs, p-CREB, and p-CaMKII was also reversed by D1DR, D2DR, and D1–D2DR complex agonists. In conclusion, these results indicate that D1DR and D2DR form a complex and in turn couple with the Gαq protein to increase neuronal excitability via PKC γ, CaMKII, MAPK, and CREB signaling in the spinal cords of CCI rats; thus, they may serve as potential drug targets for neuropathic pain therapy.Subject terms: Molecular neuroscience, Cellular neuroscience  相似文献   
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