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1.
一类具有非线性发生率和治疗函数的传染病模型研究   总被引:1,自引:0,他引:1  
传染病动力学系统的数学建模中,合理的使用非线性发生率往往更能使模型与实际相吻合.并且在实际的疾病防治过程中,由于受到空间人力物力资源的影响一般存在最大治疗容量的限制.结合这两种情况建立了一类含非线性发生率和最大治疗容量限制的传染病模型.通过分析这个模型,得到无病平衡点和正平衡点的存在性、稳定性.进一步取发生率和治疗系统达到最大容量时的感染者人数作为分支参数,得到了Hopf分支和Bogdanov-Takens分支的存在条件,并进行了数值模拟.  相似文献   

2.
传染病模型易受外界随机因素的干扰.该文提出一类具有非单调发生率的时滞随机传染病模型.利用Lyapunov方法及伊藤公式,证明了该模型具有唯一一个正全局解和该模型的无病平衡点是随机稳定的,并且得到了相应的确定型模型地方病平衡点在随机扰动下的渐近性.最后,利用数值仿真图例对理论结果加以验证说明.  相似文献   

3.
在传染病模型建模中,采用合理的非线性发生率所得到的动力学性态与实际更加接近,并且在实际的疾病防治过程中,由于受到医院各种医疗资源的影响,染病类的恢复率也会有一定的限制.建立了具有非线性发生率和恢复率函数的SIS传染病模型并分析了其动力学性态,分析这个模型,得到了无病平衡点和地方病平衡点的存在性和稳定性的条件,以及出现Hopf分支的条件.通过数值模拟,给出系统随两个分支参数变化的分支曲线图及系统的相图.  相似文献   

4.
研究一类具有非线性发生率的SIR传染病模型.应用微分方程定性理论分别得到了该系统无病平衡点、地方病平衡点全局渐近稳定的充分条件,并进行了数值模拟.  相似文献   

5.
赵英英  胡华 《应用数学》2019,32(4):796-804
本文考虑一类具有标准发生率和信息干预的时滞SIRS传染病模型.通过分析模型的特征方程,讨论无病平衡点和地方病平衡点局部渐近稳定性.应用Halanay不等式对无病平衡点的全局渐近稳定性进行证明.通过构造适当的Lyapunov函数讨论地方病平衡点全局渐近稳定性.最后通过数值模拟分析一些重要参数对疾病传播的影响.  相似文献   

6.
马霞  周义仓 《应用数学》2017,30(4):715-725
本文研究一类离散SCIRS模型的动力学性态,利用再生矩阵的方法得到模型的基本再生数,证明模型无病平衡点的全局渐近稳定性,以及模型地方病平衡点的存在性与一致持续性.数值模拟显示地方病平衡点可能是全局渐近稳定的.最后,把模型应用到我国流脑的传播中,通过数值模拟的结果和法定传染病报告的结果对比,表明该模型在一定程度上可以用来预测流脑在我国的传播.  相似文献   

7.
李洁  贾建文 《应用数学》2015,28(2):339-348
本文研究一类具有饱和传染率的SIVS传染病模型.首先利用Routh-Hurwitz判据和特征根方法,得到平衡点的局部渐近稳定性,其次证明系统的持久性和无病平衡点的全局渐近稳定性,并利用极限系统理论得到地方病平衡点的全局渐近稳定性.最后用数值模拟验证理论结果的正确性.  相似文献   

8.
主要研究了具有标准发生率和因病死亡率的离散SIS传染病模型的动力学性质,利用构造Lyapunov函数,得到模型无病平衡点和地方性平衡点的全局稳定性,即无病平衡点是全局渐近稳定的当且仅当基本再生数R_0≤1,地方病平衡点是全局渐近稳定的当且仅当R_0>1.  相似文献   

9.
研究了一类分数阶复值SIR传染病模型的稳定性.首先把原复值系统分解成实部系统和虚部系统,并讨论系统的无病平衡点.然后基于Jacobian矩阵计算出系统矩阵的特征值的正负来判断系统无病平衡点的局部稳定性,以及利用FV-1方法计算出系统的基本再生数,分析无病平衡点的全局稳定性.最后通过仿真验证了理论结果的正确性.  相似文献   

10.
穆宇光  徐瑞 《应用数学》2019,32(3):570-580
本文研究一类具有饱和发生率和复发的随机SIRI传染病模型.首先,我们证明随机系统存在唯一的全局正解.然后讨论无病平衡点的稳定性,并利用Lyapunov函数法证明流行病的灭绝.随后,我们得到疾病持久性的充分条件.最后,通过数值模拟说明结论的正确性.  相似文献   

11.
In this paper, the dynamical behavior of a virus dynamics model with CTL immune response and time delay is studied. Time delay is used to describe the time between the infected cell and the emission of viral particles on a cellular level. The effect of time delay on stability of the equilibria of the CTL immune response model has been studied and sufficient criteria for local asymptotic stability of the disease-free equilibrium, immune-free equilibrium and endemic equilibrium and global asymptotic stability of the disease-free equilibrium are given. Some conditions for Hopf bifurcation around immune-free equilibrium and endemic equilibrium to occur are also obtained by using the time delay as a bifurcation parameter. Numerical simulation with some hypothetical sets of data has been done to support the analytical findings.  相似文献   

12.
In this paper, an eco-epidemiological model with diseases in the predator and Holling type-III functional response is analyzed. A time delay due to the gestation of the predator is considered in this model. By analyzing the corresponding characteristic equations, the local stability of each of feasible equilibria and the existence of Hopf bifurcations at the disease-free equilibrium and the endemic-coexistence equilibrium are established respectively. By using Lyapunov functionals and LaSalle''s invariance principle, sufficient conditions are obtained for the global stability of the predator-extinction equilibrium, the disease-free equilibrium and the endemic-coexistence equilibrium respectively. Finally, numerical simulations are performed to illustrate the theoretical results.  相似文献   

13.
研究一类种群有迁移的流行病模型,得到了这类模型的基本再生数R0,证明了R0<1无病平衡点是局部渐近稳定的,而当R0>1时无病平衡点是不稳定的.进一步讨论了疾病持续存在与无病平衡点和地方病平衡点全局稳定的条件.  相似文献   

14.
研究了一类具有阶段结构的SIR传染病模型,在模型中假设种群分幼年和成年两个阶段,且只有成年种群染病,并且采用与成年易感者数量有关的一般非线性传染率,得到了系统解的有界性及无病平衡点和地方病平衡点存在的条件.通过对平衡点对应的特征方程的讨论得到了平衡点局部渐近稳定的条件,同时证明了平衡点的全局渐近稳定性,并对结论进行了数值模拟.  相似文献   

15.
媒体报道对疾病的预防和控制有着重要的作用,其可以减少人们感染疾病的机会.通过建立具有媒体饱和的传染病时滞模型来刻画媒体报道对感染率的影响,首先计算出无病平衡点和当R_01时存在唯一的地方病平衡点;其次,分析了平衡点的稳定性,并得到当参数满足一定条件时,时滞τ超过临界值τ_0,地方病平衡点处会出现Hopf分支;最后,通过数值模拟来验证理论分析.  相似文献   

16.
In this paper, an eco-epidemiological predator–prey model with stage structure for the prey and a time delay describing the latent period of the disease is investigated. By analyzing corresponding characteristic equations, the local stability of the trivial equilibrium, the predator-extinction equilibrium, the disease-free equilibrium and the endemic equilibrium is addressed. The existence of Hopf bifurcations at the endemic equilibrium is established. By using Lyapunov functionals and LaSalle’s invariance principle, sufficient conditions are obtained for the global asymptotic stability of the trivial equilibrium, the predator-extinction equilibrium, the disease-free equilibrium and the endemic equilibrium of the model.  相似文献   

17.
研究建立了狗与人两个群体的SEIR狂犬病模型,把狂犬病潜伏期阶段的传染性加入到模型中,利用遗传算法对1996年到2015年的数据进行拟合,估出了中国狂犬病的基本再生数R0≈1.66,证明了狂犬病无病平衡点的全局稳定性以及模型的一致持续性.根据数值模拟结果,发现处于潜伏期的犬类对人类狂犬病造成威胁.因此控制狂犬病的一个关键措施就是要做好犬的管理,减少与犬只的接触,这对抑制狂犬病的爆发有着重要的作用,也为我国狂犬病的控制提供了可靠的理论依据.  相似文献   

18.
In this paper, a delay cholera model with constant infectious period is investigated. By analyzing the characteristic equations, the local stability of a disease-free equilibrium and an endemic equilibrium of the model is established. It is proved that if the basic reproductive number $\mathcal{R}_0>1$, the system is permanent. If $\mathcal{R}_0<1$, by means of an iteration technique, sufficient conditions are obtained for the global asymptotic stability of the disease-free equilibrium. If $\mathcal{R}_0>1$, also by means of an iteration technique, sufficient conditions are obtained for the global asymptotic stability of the endemic equilibrium. Numerical simulations are carried out to illustrate the main theoretical results.  相似文献   

19.
This paper deals with prey-predator system where the prey population is infected with a microparasite. Predator functional response is assumed to be Holling type I. All the feasible equilibrium points of the system are obtained and the condition for the existence of positive (interior) equilibrium point is also determined. The criteria for both local stability and instability involving system parameters are derived. The criterion for existence of Hopf-type small periodic oscillation is shown. The condition for survival of all the population is investigated. We have developed a necessary and sufficient criteria for uniform persistence. Finally, the numerical verifications of analytic results are done.  相似文献   

20.
In this paper, a multi-scale mathematical model for environmentally transmitted diseases is proposed which couples the pathogen-immune interaction inside the human body with the disease transmission at the population level. The model is based on the nested approach that incorporates the infection-age-structured immunological dynamics into an epidemiological system structured by the chronological time, the infection age and the vaccination age. We conduct detailed analysis for both the within-host and between-host disease dynamics. Particularly, we derive the basic reproduction number R0 for the between-host model and prove the uniform persistence of the system. Furthermore, using carefully constructed Lyapunov functions, we establish threshold-type results regarding the global dynamics of the between-host system: the disease-free equilibrium is globally asymptotically stable when R0 < 1, and the endemic equilibrium is globally asymptotically stable when R0 > 1. We explore the connection between the within-host and between-host dynamics through both mathematical analysis and numerical simulation. We show that the pathogen load and immune strength at the individual level contribute to the disease transmission and spread at the population level. We also find that, although the between-host transmission risk correlates positively with the within-host pathogen load, there is no simple monotonic relationship between the disease prevalence and the individual pathogen load.  相似文献   

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